{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2025,10,19]],"date-time":"2025-10-19T15:26:50Z","timestamp":1760887610203,"version":"3.44.0"},"reference-count":50,"publisher":"BMJ","issue":"suppl 1","content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Gut"],"published-print":{"date-parts":[[2005,3]]},"abstract":"<jats:p>Barrett\u2019s oesophagus is defined as the replacement of squamous oesophageal epithelium by intestinal metaplasia in the distal oesophagus. It is a fairly frequent complication of gastro-oesophageal reflux disease (GORD): 5\u201310% of patients with GORD suffer from Barrett\u2019s oesophagus. GORD is essential for the development of Barrett\u2019s oesophagus.1 Intestinal metaplasia is a premalignant lesion that may further develop into dysplasia and lead to adenocarcinoma of the oesophagus.2 The latter now accounts for almost 50% of oesophageal cancer cases in western countries, and the largest increase in its incidence was recorded during the past two decades.3 Patients with Barrett\u2019s oesophagus have a 2\u201325% risk of developing mild to severe dysplasia and a 2\u20135% risk of having adenocarcinoma: 30\u2013150 times higher than the risk in the general population. Forty to fifty per cent of Barrett\u2019s oesophagus patients with severe dysplasia would present adenocarcinoma within 5 years.4,5<\/jats:p>","DOI":"10.1136\/gut.2004.041574","type":"journal-article","created":{"date-parts":[[2005,2,11]],"date-time":"2005-02-11T19:02:38Z","timestamp":1108148558000},"page":"i33-i37","source":"Crossref","is-referenced-by-count":8,"title":["Barrett\u2019s oesophagus: the new endoscopic modalities have a 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