{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,1,19]],"date-time":"2026-01-19T01:00:39Z","timestamp":1768784439092,"version":"3.49.0"},"reference-count":39,"publisher":"BMJ","issue":"5","content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Heart"],"published-print":{"date-parts":[[2005,5]]},"abstract":"<jats:p>\n                  <jats:bold>Objective:<\/jats:bold> To assess ethnic differences in the prevalence and aetiology of left ventricular systolic dysfunction (LVSD) in the community.<\/jats:p>\n               <jats:p>\n                  <jats:bold>Design:<\/jats:bold> Community cohort study. All patients underwent echocardiography and those found to have LVSD underwent myocardial perfusion imaging with or without coronary angiography to diagnose underlying coronary artery disease (CAD).<\/jats:p>\n               <jats:p>\n                  <jats:bold>Setting:<\/jats:bold> Seven representative general practices in Harrow, UK, a community hospital, and a local district general hospital.<\/jats:p>\n               <jats:p>\n                  <jats:bold>Patients:<\/jats:bold> 1392 patients \u2a7e 45 years old randomly selected from the computer records of seven general practices.<\/jats:p>\n               <jats:p>\n                  <jats:bold>Main outcome measures:<\/jats:bold> The prevalence and aetiology of LVSD in the community, assessing differences between white and non-white populations, and the proportion of patients with LVSD with undiagnosed CAD.<\/jats:p>\n               <jats:p>\n                  <jats:bold>Results:<\/jats:bold> 734 patients (53%) attended, 518 (71%) white and 216 (29%) non-white, the majority South Asian. Thirty nine patients (5.5%) had probable LVSD and 25 (3.5%) definite LVSD. No significant differences in prevalence were seen with ethnicity. CAD underlay most cases of LVSD. Non-white patients had a higher prevalence of CAD as the underlying aetiology of significant LVSD than white patients (100% <jats:italic>v<\/jats:italic> 56%, p \u200a=\u200a 0.04) and a trend towards less alcoholic cardiomyopathy. 8% of patients with LVSD had undiagnosed CAD.<\/jats:p>\n               <jats:p>\n                  <jats:bold>Conclusions:<\/jats:bold> LVSD is common. White and non-white patients have a similar overall prevalence of LVSD. Non-white patients, the majority South Asians in this study, have a higher prevalence of CAD as the underlying cause for LVSD than white patients. CAD underlies most cases of LVSD in the community, although it may be undiagnosed unless formally assessed.<\/jats:p>","DOI":"10.1136\/hrt.2003.029959","type":"journal-article","created":{"date-parts":[[2005,4,14]],"date-time":"2005-04-14T19:13:07Z","timestamp":1113505987000},"page":"595-600","source":"Crossref","is-referenced-by-count":27,"title":["Ethnic differences in the prevalence and aetiology of left ventricular systolic dysfunction in the community: the Harrow heart failure watch"],"prefix":"10.1136","volume":"91","author":[{"given":"G I W","family":"Galasko","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"R","family":"Senior","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"A","family":"Lahiri","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"239","published-online":{"date-parts":[[2005,4,14]]},"reference":[{"key":"2025091923355210000_91.5.595.1","first-page":"P9","volume":"19","year":"1998","journal-title":"Eur Heart J"},{"key":"2025091923355210000_91.5.595.2","doi-asserted-by":"crossref","first-page":"842","DOI":"10.1093\/eurheartj\/19.6.842","volume":"19","year":"1998","journal-title":"Eur Heart J"},{"key":"2025091923355210000_91.5.595.3","doi-asserted-by":"publisher","DOI":"10.1016\/S0140-6736(03)13501-5"},{"key":"2025091923355210000_91.5.595.4","doi-asserted-by":"publisher","DOI":"10.1016\/S0140-6736(97)03033-X"},{"key":"2025091923355210000_91.5.595.5","doi-asserted-by":"publisher","DOI":"10.1016\/S0140-6736(01)05620-3"},{"key":"2025091923355210000_91.5.595.6","doi-asserted-by":"publisher","DOI":"10.1053\/euhj.1998.1239"},{"key":"2025091923355210000_91.5.595.7","first-page":"372","volume":"351","year":"1998","journal-title":"Lancet"},{"key":"2025091923355210000_91.5.595.8","doi-asserted-by":"crossref","first-page":"368","DOI":"10.1136\/bmj.318.7180.368","volume":"318","year":"1999","journal-title":"BMJ"},{"key":"2025091923355210000_91.5.595.9","doi-asserted-by":"publisher","DOI":"10.1053\/euhj.2000.2284"},{"key":"2025091923355210000_91.5.595.10","doi-asserted-by":"publisher","DOI":"10.1067\/mhj.2001.113223"},{"key":"2025091923355210000_91.5.595.11","doi-asserted-by":"publisher","DOI":"10.1016\/S0735-1097(02)01726-6"},{"key":"2025091923355210000_91.5.595.12","unstructured":"Galasko GIW, Lahiri A. 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