{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,8,25]],"date-time":"2026-08-25T19:27:40Z","timestamp":1787686060704,"version":"build-2784847793"},"reference-count":22,"publisher":"BMJ","issue":"2","license":[{"start":{"date-parts":[[2020,6,17]],"date-time":"2020-06-17T00:00:00Z","timestamp":1592352000000},"content-version":"vor","delay-in-days":16,"URL":"http:\/\/creativecommons.org\/licenses\/by-nc\/4.0\/"}],"content-domain":{"domain":["bmj.com"],"crossmark-restriction":true},"short-container-title":["RMD Open"],"accepted":{"date-parts":[[2020,6,12]]},"published-print":{"date-parts":[[2020,6]]},"abstract":"<jats:p>A few weeks after the peak of the global 2019\nnovel coronavirus disease pandemic, cases of shock, multisystem inflammation and severe myocarditis have occurred in children and adolescents, generating some concerns and above all many questions. An almost immediate association raised with shock syndrome related to Kawasaki disease (KD). However, in light of bo\/th experience and literature have taught us about severe\nacute respiratory syndrome coronavirus 2 (SARS-COV-2) infection, and what already known on the epidemiology of KD, we suggest here the hypothesis of a new \u2018post-viral\u2019 systemic inflammatory disease related to excessive adaptive immune response rather than a form of KD caused by SARS-COV-2. We discuss analogies and differences between the two forms.<\/jats:p>","DOI":"10.1136\/rmdopen-2020-001333","type":"journal-article","created":{"date-parts":[[2020,7,1]],"date-time":"2020-07-01T14:25:13Z","timestamp":1593613513000},"page":"e001333","update-policy":"https:\/\/doi.org\/10.1136\/crossmarkpolicy","source":"Crossref","is-referenced-by-count":30,"title":["Is it Kawasaki shock syndrome, Kawasaki-like disease or pediatric inflammatory multisystem disease? 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