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Here, activation of the microdomain-confined signaling pathway with an agonist for the eNOS-coupled \u03b23-AR eliminates inhibitory glutathionylation of the Na\n                    <jats:sup>+<\/jats:sup>\n                    -K\n                    <jats:sup>+<\/jats:sup>\n                    pump\u2019s \u03b21 subunit, uncoupling of eNOS, and activation of NADPH oxidase that are sources of H\/R-induced oxidative stress. The agonist also eliminates H\/R-induced inhibition of trophoblast migration and their integration into an endothelial network.\n                  <\/jats:p>","DOI":"10.1152\/ajpcell.00379.2024","type":"journal-article","created":{"date-parts":[[2024,11,4]],"date-time":"2024-11-04T11:00:32Z","timestamp":1730718032000},"page":"C27-C39","update-policy":"https:\/\/doi.org\/10.1152\/aps-crossmark-policy","source":"Crossref","is-referenced-by-count":5,"title":["\u03b23-adrenergic agonist counters oxidative stress and Na\n                    <sup>+<\/sup>\n                    -K\n                    <sup>+<\/sup>\n                    pump inhibitory S-glutathionylation of placental cells: implications for preeclampsia"],"prefix":"10.1152","volume":"328","author":[{"ORCID":"https:\/\/orcid.org\/0000-0003-0801-5234","authenticated-orcid":false,"given":"Chia-Chi","family":"Liu","sequence":"first","affiliation":[{"name":"Kolling Medical Research Institute, University of 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