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We show that these channels are expressed in muscle afferents and generate currents that are blocked by XE991 and bradykinin (BK). The XE991 block suggests that K\n                    <jats:sub>V<\/jats:sub>\n                    7 current comprises K\n                    <jats:sub>V<\/jats:sub>\n                    7.2\/3 and K\n                    <jats:sub>V<\/jats:sub>\n                    7.5 channels. The BK inhibition of K\n                    <jats:sub>V<\/jats:sub>\n                    7 channels may explain how BK activates the exercise pressor reflex (EPR). Retigabine can enhance K\n                    <jats:sub>V<\/jats:sub>\n                    7 current, which could help control the inappropriately activated EPR in patients with cardiovascular disease.\n                  <\/jats:p>","DOI":"10.1152\/jn.00700.2020","type":"journal-article","created":{"date-parts":[[2021,5,26]],"date-time":"2021-05-26T12:10:16Z","timestamp":1622031016000},"page":"1-10","update-policy":"https:\/\/doi.org\/10.1152\/aps-crossmark-policy","source":"Crossref","is-referenced-by-count":5,"title":["K\n                    <sub>V<\/sub>\n                    7 channels are potential regulators of the exercise pressor reflex"],"prefix":"10.1152","volume":"126","author":[{"given":"Andrew B.","family":"Wright","sequence":"first","affiliation":[{"name":"The Baker Laboratory of Pharmacology, Department of Pharmacology, Kirksville College     of Osteopathic Medicine, A.T. 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