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However, the neurobiological mechanism of IAD has not bee fully expressed. The present study was designed to determine if the striatal dopamine transporter (DAT) levels measured by<mml:math xmlns:mml=\"http:\/\/www.w3.org\/1998\/Math\/MathML\"><mml:mrow><mml:mtext>T<\/mml:mtext><mml:mrow><mml:mn>99<\/mml:mn><mml:mtext>m<\/mml:mtext><\/mml:mrow><mml:mtext>c<\/mml:mtext><\/mml:mrow><\/mml:math>-TRODAT-1 single photon emission computed tomography (SPECT) brain scans were altered in individuals with IAD. SPECT brain scans were acquired on 5 male IAD subjects and 9 healthy age-matched controls. The volume (<jats:italic>V<\/jats:italic>) and weight (<jats:italic>W<\/jats:italic>) of bilateral corpus striatum as well as the<mml:math xmlns:mml=\"http:\/\/www.w3.org\/1998\/Math\/MathML\"><mml:mrow><mml:mtext>T<\/mml:mtext><mml:mrow><mml:mn>99<\/mml:mn><mml:mtext>m<\/mml:mtext><\/mml:mrow><mml:mtext>c<\/mml:mtext><\/mml:mrow><\/mml:math>-TRODAT-1 uptake ratio of corpus striatum\/the whole brain (Ra) were calculated using mathematical models. It was displayed that DAT expression level of striatum was significantly decreased and the<jats:italic>V<\/jats:italic>,<jats:italic>W<\/jats:italic>, and Ra were greatly reduced in the individuals with IAD compared to controls. Taken together, these results suggest that IAD may cause serious damages to the brain and the neuroimaging findings further illustrate IAD is associated with dysfunctions in the dopaminergic brain systems. 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