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The LPS induces inflammation by interacting with robust cellular mechanism which was considered as a plethora of numerous reproductive disorders such as fetal resorption, preterm birth, teratogenicity, intrauterine growth restriction, abortion, neural tube defects, fetal demise, and skeletal development retardation. LPS\u2010triggered overproduction of free radicals leads to oxidative stress which mediates inflammation via stimulation of NF\u2010<jats:italic>\u03ba<\/jats:italic>B and PPAR<jats:italic>\u03b3<\/jats:italic> transcription factors. Flavonoids, which exist in copious amounts in nature, possess a wide array of functions; their supplementation during pregnancy activates Nrf2 signaling pathway which encounters pregnancy disorders. It was further presumed that the development of strong antioxidant uterine environment during gestation can alleviate diseases which appear at adult stages. 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