{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2025,12,31]],"date-time":"2025-12-31T07:25:40Z","timestamp":1767165940627,"version":"build-2238731810"},"reference-count":31,"publisher":"Wiley","issue":"1","license":[{"start":{"date-parts":[[2022,8,18]],"date-time":"2022-08-18T00:00:00Z","timestamp":1660780800000},"content-version":"vor","delay-in-days":229,"URL":"http:\/\/creativecommons.org\/licenses\/by\/4.0\/"}],"funder":[{"DOI":"10.13039\/501100001809","name":"National Natural Science Foundation of China","doi-asserted-by":"publisher","award":["81402629"],"award-info":[{"award-number":["81402629"]}],"id":[{"id":"10.13039\/501100001809","id-type":"DOI","asserted-by":"publisher"}]}],"content-domain":{"domain":["onlinelibrary.wiley.com"],"crossmark-restriction":true},"short-container-title":["Oxidative Medicine and Cellular Longevity"],"published-print":{"date-parts":[[2022,1]]},"abstract":"<jats:p>\n                    The purpose of this study was to determine the role of heat shock protein 72 (Hsp72) changes in cardiac injury caused by microwave radiation, aimed at providing novel insights into the mechanism of this damage. A digital thermometer was used to measure the rectal temperature of the rats\u2019 pre\u2010 and post\u2010radiation. On the 1\n                    <jats:sup>st<\/jats:sup>\n                    , 7\n                    <jats:sup>th<\/jats:sup>\n                    , 14\n                    <jats:sup>th<\/jats:sup>\n                    , and 28\n                    <jats:sup>th<\/jats:sup>\n                    days post\u2010radiation, the changes in electrocardiogram (ECG) were analyzed by a multi\u2010channel physiological recorder. The myocardial enzyme activities and ion concentrations were detected by an automatic biochemical analyzer. Additionally, the levels of myocardial injury markers were established by the enzyme\u2010linked immunosorbent assay (ELISA), and those of hormones were measured by radioimmunoassay. The structure and ultrastructure of the myocardial tissue were observed using an optical microscope and transmission electron microscopy (TEM). The expression of Hsp72 was measured by Western blot and immunofluorescence analyses. Post\u2010exposure, the rectal temperature in the R\u2010group increased significantly, ECG was disordered, and the concentrations of ions were decreased. Furthermore, the activities of myocardial enzymes were changed, and the contents of myocardial injury markers and hormones were increased. We observed damage to the structure and ultrastructure and significantly increased expression of Hsp72. As a whole, the results indicated that S\u2010wave microwave radiation at 30\u2009mW\/cm\n                    <jats:sup>2<\/jats:sup>\n                    for 35\u2009min resulted in damage to the cardiac functionality organigram, caused by a combination of the thermal and nonthermal effects.\n                  <\/jats:p>","DOI":"10.1155\/2022\/7145415","type":"journal-article","created":{"date-parts":[[2022,8,18]],"date-time":"2022-08-18T19:50:07Z","timestamp":1660852207000},"update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":3,"title":["[Retracted] Hsp72\u2010Based Effect and Mechanism of Microwave Radiation\u2010Induced Cardiac Injury in 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