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Although these problems occur mainly in elderly, the incidence in younger people is being reported, and an undetermined portion of patients without the classic risk factors develop subclinical atherosclerosis at earlier stages of life. Recently, both the <jats:italic>H. pylori<\/jats:italic> infection and the intestinal microbiota have been linked to atherosclerosis. The mechanisms behind those associations are poorly understood, but some of the proposed explanations are (a) the effect of the chronic systemic inflammation induced by <jats:italic>H. pylori<\/jats:italic>, (b) a direct action over the endothelial cells by the cytotoxin associated gene A protein, and (c) alterations of the lipid metabolism and endothelial dysfunction induced by <jats:italic>H. pylori<\/jats:italic> infection. Regarding the microbiota, several studies show that induction of atherosclerosis is related to high levels of Trimethylamine N\u2010oxide. In this review, we present the information published about the effects of <jats:italic>H. pylori<\/jats:italic> over the intestinal microbiota and their relationship with atherosclerosis and propose a hypothesis to explain the nature of these associations. If <jats:italic>H. pylori<\/jats:italic> contributes to atherosclerosis, then interventions for eradication and restoration of the gut microbiota at early stages could represent a way to prevent disease progression.<\/jats:p>","DOI":"10.1155\/2022\/9943158","type":"journal-article","created":{"date-parts":[[2022,10,23]],"date-time":"2022-10-23T05:20:06Z","timestamp":1666502406000},"update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":18,"title":["<i>Helicobacter Pylori<\/i> Infection Induces Intestinal Dysbiosis That Could Be Related to the Onset of 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