{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,10,7]],"date-time":"2026-10-07T00:15:57Z","timestamp":1791332157688,"version":"4.3.1"},"reference-count":10,"publisher":"Bioscientifica","issue":"3","license":[{"start":{"date-parts":[[2026,2,16]],"date-time":"2026-02-16T00:00:00Z","timestamp":1771200000000},"content-version":"vor","delay-in-days":3475,"URL":"https:\/\/www.karger.com\/Services\/SiteLicenses"}],"content-domain":{"domain":["journals.bioscientifica.com"],"crossmark-restriction":true},"short-container-title":[],"published-print":{"date-parts":[[2016,9,1]]},"abstract":"<jats:title>Abstract<\/jats:title>\n                  <jats:p>We describe a case of biochemical neonatal thyrotoxicosis caused by biotin supplementation. Biotin may interact with thyroid function testing to imitate thyrotoxicosis with low thyroid-stimulating hormone and elevated triiodothyronine and thyroxine levels.<\/jats:p>","DOI":"10.1159\/000448034","type":"journal-article","created":{"date-parts":[[2016,8,11]],"date-time":"2016-08-11T17:01:23Z","timestamp":1470934883000},"page":"212-215","update-policy":"https:\/\/doi.org\/10.1530\/crossmarkpolicy-17","source":"Crossref","is-referenced-by-count":22,"title":["Biochemical Hyperthyroidism in a Newborn Baby Caused by Assay Interaction from Biotin Intake"],"prefix":"10.1530","volume":"5","author":[{"given":"Inge","family":"B\u00fclow Pedersen","sequence":"first","affiliation":[{"name":"1Department of Endocrinology and Medicine, Aalborg University Hospital, Aalborg, Denmark"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Peter","family":"Laurberg","sequence":"additional","affiliation":[{"name":"1Department of Endocrinology and Medicine, Aalborg University Hospital, Aalborg, Denmark"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"416","published-online":{"date-parts":[[2016,8,12]]},"reference":[{"key":"2026070712473978600_ref1","doi-asserted-by":"crossref","unstructured":"F\u00fchrer D, Wonerow P, Willgerodt H, Paschke R: Identification of a new thyrotropin receptor germline mutation (Leu629Phe) in a family with neonatal onset of autosomal dominant nonautoimmune hyperthyroidism. 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