{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,1,13]],"date-time":"2026-01-13T13:52:12Z","timestamp":1768312332225,"version":"3.49.0"},"reference-count":0,"publisher":"Ovid Technologies (Wolters Kluwer Health)","issue":"12","content-domain":{"domain":["www.ahajournals.org"],"crossmark-restriction":true},"short-container-title":["Stroke"],"published-print":{"date-parts":[[1989,12]]},"abstract":"<jats:p>We reviewed 49 patients with Wernicke's aphasia resulting from a stroke. Their aphasia was classified on the basis of comprehensive neuropsychological testing. Wernicke's aphasia was more common in older patients and in men. Cerebral infarction occurred in 38 patients (78%) and intracerebral hemorrhage in seven (14%); the remaining four patients (8%) developed aphasia after surgery for aneurysmal subarachnoid hemorrhage. Embolic events were the most common etiology of Wernicke's aphasia in the 38 patients with cerebral infarction, with cardiac emboli in 40% and large-vessel atheroemboli from a carotid source in 16%. In patients with Wernicke's aphasia secondary to infarction, an embolic source should be sought. Patients with Wernicke's aphasia should have computed tomography to exclude intracerebral hemorrhage before institution of anticoagulant therapy.<\/jats:p>","DOI":"10.1161\/01.str.20.12.1730","type":"journal-article","created":{"date-parts":[[2011,6,17]],"date-time":"2011-06-17T20:08:03Z","timestamp":1308341283000},"page":"1730-1732","update-policy":"https:\/\/doi.org\/10.1161\/crossmarkpolicy","source":"Crossref","is-referenced-by-count":23,"title":["Etiology of stroke in patients with Wernicke's aphasia."],"prefix":"10.1161","volume":"20","author":[{"given":"L E","family":"Knepper","sequence":"first","affiliation":[{"name":"Division of Cerebrovascular Disease, University of Iowa College of Medicine, Iowa City."}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"J","family":"Biller","sequence":"additional","affiliation":[{"name":"Division of Cerebrovascular Disease, University of Iowa College of Medicine, Iowa City."}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"D","family":"Tranel","sequence":"additional","affiliation":[{"name":"Division of Cerebrovascular Disease, University of Iowa College of Medicine, Iowa City."}],"role":[{"role":"author","vocabulary":"crossref"}]},{"suffix":"Jr","given":"H P","family":"Adams","sequence":"additional","affiliation":[{"name":"Division of Cerebrovascular Disease, University of Iowa College of Medicine, Iowa City."}],"role":[{"role":"author","vocabulary":"crossref"}]},{"suffix":"3rd","given":"E E","family":"Marsh","sequence":"additional","affiliation":[{"name":"Division of Cerebrovascular Disease, University of Iowa College of Medicine, Iowa City."}],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"276","container-title":["Stroke"],"original-title":[],"language":"en","link":[{"URL":"https:\/\/www.ahajournals.org\/doi\/pdf\/10.1161\/01.STR.20.12.1730","content-type":"unspecified","content-version":"vor","intended-application":"similarity-checking"}],"deposited":{"date-parts":[[2024,5,12]],"date-time":"2024-05-12T21:39:44Z","timestamp":1715549984000},"score":1,"resource":{"primary":{"URL":"https:\/\/www.ahajournals.org\/doi\/10.1161\/01.STR.20.12.1730"}},"subtitle":[],"short-title":[],"issued":{"date-parts":[[1989,12]]},"references-count":0,"journal-issue":{"issue":"12","published-print":{"date-parts":[[1989,12]]}},"alternative-id":["10.1161\/01.STR.20.12.1730"],"URL":"https:\/\/doi.org\/10.1161\/01.str.20.12.1730","relation":{},"ISSN":["0039-2499","1524-4628"],"issn-type":[{"value":"0039-2499","type":"print"},{"value":"1524-4628","type":"electronic"}],"subject":[],"published":{"date-parts":[[1989,12]]},"assertion":[{"value":"1989-12-01","order":2,"name":"published","label":"Published","group":{"name":"publication_history","label":"Publication History"}}]}}