{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,10,4]],"date-time":"2026-10-04T02:47:33Z","timestamp":1791082053224,"version":"4.1.0"},"reference-count":23,"publisher":"Ovid Technologies (Wolters Kluwer Health)","issue":"1","content-domain":{"domain":["www.ahajournals.org"],"crossmark-restriction":true},"short-container-title":["Stroke"],"published-print":{"date-parts":[[1995,1]]},"abstract":"<jats:p>\n            <jats:italic>Background<\/jats:italic>\n            Elevated transcranial Doppler (TCD) velocities seen after cerebral angioplasty are commonly interpreted as evidence of residual or recurrent stenosis but may conceivably arise from hyperemia and require different clinical management.\n          <\/jats:p>\n          <jats:p>\n            <jats:italic>Summary of Report<\/jats:italic>\n            Four cases of abnormally elevated mean TCD velocities obtained after therapeutic arterial dilation with either balloon angioplasty or intra-arterial administration of papaverine are described. In each case, cerebral angiography revealed a dilated vessel, suggesting that hyperemia and impaired autoregulation were the causes of the high velocities.\n          <\/jats:p>\n          <jats:p>\n            <jats:italic>Conclusions<\/jats:italic>\n            These examples suggest that high TCD velocities after vessel dilation may be produced by unpredictable amounts of vessel narrowing and flow alteration. Although a normalizing TCD velocity after angioplasty suggests effective vessel dilation, high velocities may be due partly to hyperemia and cannot be interpreted as arising solely from recurrent stenosis.\n          <\/jats:p>","DOI":"10.1161\/01.str.26.1.123","type":"journal-article","created":{"date-parts":[[2011,6,17]],"date-time":"2011-06-17T20:08:03Z","timestamp":1308341283000},"page":"123-127","update-policy":"https:\/\/doi.org\/10.1161\/crossmarkpolicy","source":"Crossref","is-referenced-by-count":20,"title":["Elevated Transcranial Doppler Ultrasound Velocities Following Therapeutic Arterial Dilation"],"prefix":"10.1161","volume":"26","author":[{"given":"Cole A.","family":"Giller","sequence":"first","affiliation":[{"name":"From the Departments of Neurosurgery (C.A.G., A.G., H.H.B., T.K.) and Radiology (C.A.G., P.P.), University of Texas Southwestern Medical Center at Dallas."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Phil","family":"Purdy","sequence":"additional","affiliation":[{"name":"From the Departments of Neurosurgery (C.A.G., A.G., H.H.B., T.K.) and Radiology (C.A.G., P.P.), University of Texas Southwestern Medical Center at Dallas."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Angela","family":"Giller","sequence":"additional","affiliation":[{"name":"From the Departments of Neurosurgery (C.A.G., A.G., H.H.B., T.K.) and Radiology (C.A.G., P.P.), University of Texas Southwestern Medical Center at Dallas."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"H. Hunt","family":"Batjer","sequence":"additional","affiliation":[{"name":"From the Departments of Neurosurgery (C.A.G., A.G., H.H.B., T.K.) and Radiology (C.A.G., P.P.), University of Texas Southwestern Medical Center at Dallas."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Tom","family":"Kopitnik","sequence":"additional","affiliation":[{"name":"From the Departments of Neurosurgery (C.A.G., A.G., H.H.B., T.K.) and Radiology (C.A.G., P.P.), University of Texas Southwestern Medical Center at Dallas."}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"276","reference":[{"key":"e_1_3_2_1_2","first-page":"81","volume":"42","year":"1984","unstructured":"Aaslid R, Huber P, Nornes H. Evaluation of cerebrovascular spasm with transcranial Doppler ultrasound. 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