{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,10,9]],"date-time":"2026-10-09T21:59:41Z","timestamp":1791583181008,"version":"4.3.4"},"reference-count":159,"publisher":"Ovid Technologies (Wolters Kluwer Health)","issue":"9","content-domain":{"domain":["www.ahajournals.org"],"crossmark-restriction":true},"short-container-title":["ATVB"],"published-print":{"date-parts":[[2020,9]]},"abstract":"<jats:p>\n            The world is amid a pandemic caused by severe acute respiratory syndrome-coronavirus 2. Severe acute respiratory syndrome-coronavirus causes serious respiratory tract infections that can lead to viral pneumonia, acute respiratory distress syndrome, and death. Some patients with coronavirus disease 2019 (COVID-19) have an activated coagulation system characterized by elevated plasma levels of\n            <jats:sc>d<\/jats:sc>\n            -dimer\u2014a biomarker of fibrin degradation. Importantly, high levels of\n            <jats:sc>D<\/jats:sc>\n            -dimer on hospital admission are associated with increased risk of mortality. Venous thromboembolism is more common than arterial thromboembolism in hospitalized COVID-19 patients. Pulmonary thrombosis and microvascular thrombosis are observed in autopsy studies, and this may contribute to the severe hypoxia observed in COVID-19 patients. It is likely that multiple systems contribute to thrombosis in COVID-19 patients, such as activation of coagulation, platelet activation, hypofibrinolysis, endothelial cell dysfunction, inflammation, neutrophil extracellular traps, and complement. Targeting these different pathways may reduce thrombosis and improve lung function in COVID-19 patients.\n          <\/jats:p>","DOI":"10.1161\/atvbaha.120.314514","type":"journal-article","created":{"date-parts":[[2020,7,13]],"date-time":"2020-07-13T13:30:27Z","timestamp":1594647027000},"page":"2033-2044","update-policy":"https:\/\/doi.org\/10.1161\/crossmarkpolicy","source":"Crossref","is-referenced-by-count":172,"title":["Coagulation Abnormalities and Thrombosis in Patients Infected With SARS-CoV-2 and Other Pandemic Viruses"],"prefix":"10.1161","volume":"40","author":[{"given":"Nigel","family":"Mackman","sequence":"first","affiliation":[{"name":"From the Department of Medicine, UNC Blood Research Center (N.M., S.A., A.S.W., R.K., N.S.K.), University of North Carolina at Chapel Hill."},{"name":"Division of Hematology, Department of Medicine (N.M., R.K., N.S.K.), University of North Carolina at Chapel 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