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HNO increased the\n            <jats:italic>S<\/jats:italic>\n            -glutathiolation of SERCA, and adenoviral overexpression of glutaredoxin-1 prevented both the HNO-stimulated oxidative modification of SERCA and its activation, as did overexpression of a mutated SERCA in which cysteine 674 was replaced with serine. Thus, HNO increases the maximal activation of SERCA via\n            <jats:italic>S<\/jats:italic>\n            -glutathiolation at cysteine 674.\n          <\/jats:p>","DOI":"10.1161\/circresaha.108.188441","type":"journal-article","created":{"date-parts":[[2009,3,6]],"date-time":"2009-03-06T05:09:14Z","timestamp":1236316154000},"page":"720-723","update-policy":"https:\/\/doi.org\/10.1161\/crossmarkpolicy","source":"Crossref","is-referenced-by-count":145,"title":["Nitroxyl Activates SERCA in Cardiac Myocytes via Glutathiolation of Cysteine 674"],"prefix":"10.1161","volume":"104","author":[{"given":"Steve","family":"Lancel","sequence":"first","affiliation":[{"name":"From the Cardiovascular Medicine Section, Department of Medicine, and the Myocardial and Vascular Biology Units, Boston University Medical Center, Mass."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Jingmei","family":"Zhang","sequence":"additional","affiliation":[{"name":"From the Cardiovascular Medicine 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