{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,10,4]],"date-time":"2026-10-04T21:45:58Z","timestamp":1791150358206,"version":"4.1.0"},"reference-count":27,"publisher":"Ovid Technologies (Wolters Kluwer Health)","issue":"15","content-domain":{"domain":["www.ahajournals.org"],"crossmark-restriction":true},"short-container-title":["Circulation"],"published-print":{"date-parts":[[2009,10,13]]},"abstract":"<jats:p>\n            <jats:bold>\n              <jats:italic>Background\u2014<\/jats:italic>\n            <\/jats:bold>\n            Currently, only anecdotal information exists on the presentation and outcome of coronary arterial injury after ablation procedures.\n          <\/jats:p>\n          <jats:p>\n            <jats:bold>\n              <jats:italic>Methods and Results\u2014<\/jats:italic>\n            <\/jats:bold>\n            Four patients who sustained coronary artery injury of a cohort of patients undergoing 4655 consecutive ablation procedures (0.09%) are described. The patients\u2019 mean age was 45\u00b111 years, and 1.8\u00b10.5 prior ablation attempts had been unsuccessful. Coronary injury occurred from epicardial ventricular tachycardia ablation in 2 patients (irrigated radiofrequency ablation in one and cryoablation in the other) and ablation within the middle cardiac vein with irrigated radiofrequency in 2 patients. All involved branches of the right coronary artery. Acute occlusion presenting with ST-segment elevation immediately after ablation was recognized during the procedure in 2 cases. Occlusion failed to respond to nitroglycerin or balloon dilation, and stenting was required in both cases. Acute myocardial infarction occurred 2 weeks after epicardial ablation as a result of occlusion of a right ventricular branch of the right coronary artery giving rise to the posterior descending coronary artery in 1 patient. A moderate asymptomatic stenosis was seen on angiography after epicardial cryoablation in 1 patient. All patients recovered and remained asymptomatic from the coronary injury and arrhythmias during 37\u00b153 months of follow-up.\n          <\/jats:p>\n          <jats:p>\n            <jats:bold>\n              <jats:italic>Conclusions\u2014<\/jats:italic>\n            <\/jats:bold>\n            Coronary arterial injury after ablation procedures is rare. It may present acutely or several weeks after an ablation procedure. Acute occlusion appears to require coronary stenting. Unanticipated anatomic variations can predispose to coronary injury.\n          <\/jats:p>","DOI":"10.1161\/circulationaha.109.870790","type":"journal-article","created":{"date-parts":[[2009,9,29]],"date-time":"2009-09-29T01:25:15Z","timestamp":1254187515000},"page":"1465-1473","update-policy":"https:\/\/doi.org\/10.1161\/crossmarkpolicy","source":"Crossref","is-referenced-by-count":154,"title":["Coronary Artery Injury Due to Catheter Ablation in Adults"],"prefix":"10.1161","volume":"120","author":[{"given":"Kurt C.","family":"Roberts-Thomson","sequence":"first","affiliation":[{"name":"From the Cardiac Arrhythmia Division, Cardiovascular Division, Brigham and Women\u2019s Hospital, and Harvard Medical School, Boston, Mass."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Daniel","family":"Steven","sequence":"additional","affiliation":[{"name":"From the Cardiac Arrhythmia Division, Cardiovascular Division, Brigham and Women\u2019s Hospital, and Harvard Medical School, Boston, Mass."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Jens","family":"Seiler","sequence":"additional","affiliation":[{"name":"From the Cardiac Arrhythmia Division, Cardiovascular Division, Brigham and Women\u2019s Hospital, and Harvard Medical School, Boston, Mass."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Keiichi","family":"Inada","sequence":"additional","affiliation":[{"name":"From the Cardiac Arrhythmia Division, Cardiovascular Division, Brigham and Women\u2019s Hospital, and Harvard Medical School, Boston, Mass."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Bruce A.","family":"Koplan","sequence":"additional","affiliation":[{"name":"From the Cardiac Arrhythmia Division, Cardiovascular Division, Brigham and Women\u2019s Hospital, and Harvard Medical School, Boston, Mass."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Usha B.","family":"Tedrow","sequence":"additional","affiliation":[{"name":"From the Cardiac Arrhythmia Division, Cardiovascular Division, Brigham and Women\u2019s Hospital, and Harvard Medical School, Boston, Mass."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Laurence M.","family":"Epstein","sequence":"additional","affiliation":[{"name":"From the Cardiac Arrhythmia Division, Cardiovascular Division, Brigham and Women\u2019s Hospital, and Harvard Medical School, Boston, Mass."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"William G.","family":"Stevenson","sequence":"additional","affiliation":[{"name":"From the Cardiac Arrhythmia Division, Cardiovascular Division, Brigham and Women\u2019s Hospital, and Harvard Medical School, Boston, Mass."}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"276","reference":[{"key":"e_1_3_2_1_2","doi-asserted-by":"publisher","DOI":"10.1111\/j.1540-8159.2000.tb00891.x"},{"key":"e_1_3_2_2_2","doi-asserted-by":"publisher","DOI":"10.1016\/0735-1097(92)90338-N"},{"key":"e_1_3_2_3_2","doi-asserted-by":"publisher","DOI":"10.1111\/j.1540-8167.1996.tb00559.x"},{"key":"e_1_3_2_4_2","doi-asserted-by":"publisher","DOI":"10.1093\/eurheartj\/14.12.1644"},{"key":"e_1_3_2_5_2","doi-asserted-by":"publisher","DOI":"10.1016\/S0002-9149(97)89132-1"},{"key":"e_1_3_2_6_2","doi-asserted-by":"publisher","DOI":"10.1016\/j.amjcard.2007.03.082"},{"key":"e_1_3_2_7_2","first-page":"605.","volume":"24","year":"2001","unstructured":"Sun Y, Po S, Arruda M, Scherlag B, Pitha J, Tondo C, Lazzara R, Jackman W. 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