{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,10,17]],"date-time":"2026-10-17T20:43:59Z","timestamp":1792269839858,"version":"4.4.0"},"reference-count":35,"publisher":"Oxford University Press (OUP)","issue":"1","license":[{"start":{"date-parts":[[1998,7,1]],"date-time":"1998-07-01T00:00:00Z","timestamp":899251200000},"content-version":"vor","delay-in-days":0,"URL":"https:\/\/academic.oup.com\/pages\/standard-publication-reuse-rights"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":[],"published-print":{"date-parts":[[1998,7,1]]},"abstract":"<jats:title>Abstract<\/jats:title>\n                  <jats:p>The associations between lung function measures (spirometry and peak expiratory flow lability) and estimated 20-yr ambient concentrations of respirable particles, suspended sulfates, sulfur dioxide, ozone, and indoor particles were studied in a sample of 1,391 nonsmokers followed since 1977. Differences in air pollutants across the population were associated with decrements of lung function. An increase of 54 d\/yr when particles &amp;lt; 10 \u03bc m in diameter (PM10) exceeded 100 \u03bc g\/m3 was associated with a 7.2% decrement in FEV1, as percent of predicted, in males whose parents had asthma, bronchitis, emphysema, or hay fever and with increased peak expiratory flow lability of 0.8% for all females and 0.6% for all males. An increase in mean SO4 concentration of 1.6 \u03bc g\/m3 was associated with a 1.5% decrement in FEV1, as percent of predicted, in all males. An increase of 23 ppb of ozone as an 8-h average was associated with a 6.3% decrement in FEV1, as percent of predicted, in males whose parents had asthma, bronchitis, emphysema, or hay fever.<\/jats:p>","DOI":"10.1164\/ajrccm.158.1.9710101","type":"journal-article","created":{"date-parts":[[2013,4,4]],"date-time":"2013-04-04T13:37:08Z","timestamp":1365082628000},"page":"289-298","source":"Crossref","is-referenced-by-count":143,"title":["Long-term Particulate and Other Air Pollutants and Lung Function in Nonsmokers"],"prefix":"10.1093","volume":"158","author":[{"given":"DAVID E.","family":"ABBEY","sequence":"first","affiliation":[{"name":"Loma Linda University, School of Public Health, Loma Linda, California; U.S. Environmental Protection Agency, Research Triangle Park, North Carolina; and University of Arizona Prevention Center and Respiratory Sciences Center, Tucson, Arizona"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"RAOUL J.","family":"BURCHETTE","sequence":"additional","affiliation":[{"name":"Loma Linda University, School of Public Health, Loma Linda, California; U.S. Environmental Protection Agency, Research Triangle Park, North Carolina; and University of Arizona Prevention Center and Respiratory Sciences Center, Tucson, Arizona"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"SYNN\u00d8VE F.","family":"KNUTSEN","sequence":"additional","affiliation":[{"name":"Loma Linda University, School of Public Health, Loma Linda, California; U.S. Environmental Protection Agency, Research Triangle Park, North Carolina; and University of Arizona Prevention Center and Respiratory Sciences Center, Tucson, Arizona"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"WILLIAM F.","family":"McDONNELL","sequence":"additional","affiliation":[{"name":"Loma Linda University, School of Public Health, Loma Linda, California; U.S. Environmental Protection Agency, Research Triangle Park, North Carolina; and University of Arizona Prevention Center and Respiratory Sciences Center, Tucson, Arizona"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"MICHAEL D.","family":"LEBOWITZ","sequence":"additional","affiliation":[{"name":"Loma Linda University, School of Public Health, Loma Linda, California; U.S. Environmental Protection Agency, Research Triangle Park, North Carolina; and University of Arizona Prevention Center and Respiratory Sciences Center, Tucson, Arizona"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"PAUL L.","family":"ENRIGHT","sequence":"additional","affiliation":[{"name":"Loma Linda University, School of Public Health, Loma Linda, California; U.S. Environmental Protection Agency, Research Triangle Park, North Carolina; and University of Arizona Prevention Center and Respiratory Sciences Center, Tucson, Arizona"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"286","published-online":{"date-parts":[[1998,7,1]]},"reference":[{"key":"2026032709023394700_B1","doi-asserted-by":"crossref","first-page":"350","DOI":"10.2105\/AJPH.81.3.350","article-title":"The UCLA population studies of CORD: X. 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