{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,4,29]],"date-time":"2026-04-29T00:23:39Z","timestamp":1777422219528,"version":"3.51.4"},"reference-count":23,"publisher":"SAGE Publications","issue":"1-6","license":[{"start":{"date-parts":[[1976,1,1]],"date-time":"1976-01-01T00:00:00Z","timestamp":189302400000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/journals.sagepub.com\/page\/policies\/text-and-data-mining-license"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Ann Clin Biochem"],"published-print":{"date-parts":[[1976,1]]},"abstract":"<jats:p>Hypocalcaemia accounts for many of the telephone inquiries made to the Department of Biochemical Medicine in Dundee. About half of these cases can be explained by hypoalbuminaemia; the causes of \u201ctrue\u201d hypocalcaemia are given in Table 1.<\/jats:p>\n                  <jats:p>Chronic renal failure is probably the most frequent cause of hypocalcaemia in Britain. In the world as a whole it is likely that vitamin D deficiency, causing rickets in children and osteomalacia in adults, is the commonest cause of hypocalcaemia.<\/jats:p>","DOI":"10.1177\/000456327601300162","type":"journal-article","created":{"date-parts":[[2014,2,5]],"date-time":"2014-02-05T16:56:30Z","timestamp":1391619390000},"page":"578-584","source":"Crossref","is-referenced-by-count":3,"title":["Hypocalcaemia: Differential Diagnosis and Investigation"],"prefix":"10.1177","volume":"13","author":[{"given":"C. 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