{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,5,27]],"date-time":"2026-05-27T20:56:49Z","timestamp":1779915409350,"version":"3.53.1"},"reference-count":35,"publisher":"SAGE Publications","issue":"5","license":[{"start":{"date-parts":[[2003,10,1]],"date-time":"2003-10-01T00:00:00Z","timestamp":1064966400000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/journals.sagepub.com\/page\/policies\/text-and-data-mining-license"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Behav Modif"],"published-print":{"date-parts":[[2003,10]]},"abstract":"<jats:p>\n                    Previous studies in our laboratory have shown that a single exposure to 100% carbon dioxide (CO\n                    <jats:sub>2<\/jats:sub>\n                    ) can serve as an effective unconditioned stimulus (US) in a Pavlovian aversive-context conditioning paradigm in rats. Although the US exposure parameters employed in the initial studies were sufficient for producing a context-specific enhancement of behavioral freezing and analgesia, it had yet to be determined whether variations of these CO\n                    <jats:sub>2<\/jats:sub>\n                    conditioning procedures would produce other conditioning effects. Thus, the purpose of the following experiment was to investigate the intensity of the US on the conditioned response (CR). The findings confirm that variations in CO\n                    <jats:sub>2<\/jats:sub>\n                    concentrations produce changes in the CR that are consistent with principles of Pavlovian conditioning. The findings lend additional support to the tenability of a dyspneic suffocation fear theory of panic disorder, a theory that postulates that at least one type of panic attack could be a consequence of Pavlovian conditioning.\n                  <\/jats:p>","DOI":"10.1177\/0145445503256316","type":"journal-article","created":{"date-parts":[[2003,9,20]],"date-time":"2003-09-20T04:25:16Z","timestamp":1064031916000},"page":"620-636","source":"Crossref","is-referenced-by-count":23,"title":["The Conditioning of Dyspneic Suffocation Fear"],"prefix":"10.1177","volume":"27","author":[{"given":"Donna L.","family":"Mongeluzi","sequence":"first","affiliation":[{"name":"University at Albany, State University of New York"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Robert A.","family":"Rosellini","sequence":"additional","affiliation":[{"name":"University at Albany, State University of New York"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Ronald","family":"Ley","sequence":"additional","affiliation":[{"name":"University at Albany, State University of New York"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Barbara J.","family":"Caldarone","sequence":"additional","affiliation":[{"name":"Yale University School of Medicine"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Howard S.","family":"Stock","sequence":"additional","affiliation":[{"name":"Roche Pharmaceuticals"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"179","published-online":{"date-parts":[[2003,10,1]]},"reference":[{"key":"atypb1","doi-asserted-by":"publisher","DOI":"10.1016\/0031-9384(93)90113-T"},{"key":"atypb2","doi-asserted-by":"publisher","DOI":"10.1037\/h0026779"},{"key":"atypb3","doi-asserted-by":"publisher","DOI":"10.1017\/S0140525X0000491X"},{"key":"atypb4","doi-asserted-by":"publisher","DOI":"10.1037\/h0042582"},{"key":"atypb5","unstructured":"D'Amour, F. E. & Smith, D. L. (1941). A method for determining loss of pain sensation. Journal of Pharmacology & Experimental Therapeutics, 72, 74-79."},{"key":"atypb6","doi-asserted-by":"publisher","DOI":"10.1037\/0735-7044.98.1.79"},{"key":"atypb7","doi-asserted-by":"crossref","unstructured":"Fanselow, M. S. (1991). The midbrain periaqueductal gray as a coordinator of action in response to fear and anxiety. In A. Depaulis & R. Bandler (Eds.), The midbrain periacqueductal gray matter: Functional, anatomical and neurochemical organization (pp. 151-173). 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