{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,5,4]],"date-time":"2026-05-04T18:35:23Z","timestamp":1777919723196,"version":"3.51.4"},"reference-count":20,"publisher":"SAGE Publications","issue":"2","license":[{"start":{"date-parts":[[1969,3,1]],"date-time":"1969-03-01T00:00:00Z","timestamp":-26438400000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/journals.sagepub.com\/page\/policies\/text-and-data-mining-license"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Pathologia veterinaria"],"published-print":{"date-parts":[[1969,3]]},"abstract":"<jats:p>Seven casts of generalised amyloidosis in the cat, occurring in 100 consecutive cat autopsies, were studied pathologically. Tissues from 2 of the cats were further investigated using the electron microscope. None of the cats had suffered from a prolonged infection, but one had had chronic allergic dermatitis. In 6 of the 7 cats chronic hypervitaminosis A coexisted with generalised amyloidosis. It is suggested that chronic hypervitaminosis A, which causes prolonged stimulation of the reticulo-endothelial system, may have predisposed to the high incidence of generalised amyloidosis reported in this paper.<\/jats:p>","DOI":"10.1177\/030098586900600203","type":"journal-article","created":{"date-parts":[[2010,3,8]],"date-time":"2010-03-08T17:27:48Z","timestamp":1268069268000},"page":"117-134","source":"Crossref","is-referenced-by-count":25,"title":["Generalised Amyloidosis in Seven Cats"],"prefix":"10.1177","volume":"6","author":[{"given":"Leigh","family":"Clark","sequence":"first","affiliation":[{"name":"Department of Preventive Medicine, Veterinary School, University of\r            Queensland, St. Lucia, Queensland"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"A. 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