{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,5,4]],"date-time":"2026-05-04T18:38:01Z","timestamp":1777919881470,"version":"3.51.4"},"reference-count":43,"publisher":"SAGE Publications","issue":"1","license":[{"start":{"date-parts":[[1977,1,1]],"date-time":"1977-01-01T00:00:00Z","timestamp":220924800000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/journals.sagepub.com\/page\/policies\/text-and-data-mining-license"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Vet Pathol"],"published-print":{"date-parts":[[1977,1]]},"abstract":"<jats:p>\n                    Rectal stricture occurred in each of 23 pigs submitted for necropsy from eight Indiana farms. Each stricture was an annular cicatrization of the rectal wall, 2.0-5.0 cm anterior to the anorectal junction. Emaciation, colonic dilatation and compression atrophy of abdominal and thoracic viscera were a result of chronic obstipation and inanition. Most pigs with strictures had severe enteric disease 4-8 weeks before the strictures occurred.\n                    <jats:italic toggle=\"yes\">Salmonella typhimurium<\/jats:italic>\n                    was isolated from seven of the eight groups of pigs examined. Ulcerative proctitis, a possible precursor of rectal stricture, was frequently in pigs with enterocolitis caused by 5.\n                    <jats:italic toggle=\"yes\">typhimurium<\/jats:italic>\n                    .\n                  <\/jats:p>","DOI":"10.1177\/030098587701400105","type":"journal-article","created":{"date-parts":[[2010,3,8]],"date-time":"2010-03-08T17:25:32Z","timestamp":1268069132000},"page":"36-42","source":"Crossref","is-referenced-by-count":14,"title":["The Pathogenesis of Porcine Rectal Stricture: I. 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