{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,7,23]],"date-time":"2026-07-23T18:18:39Z","timestamp":1784830719435,"version":"3.55.0"},"reference-count":24,"publisher":"SAGE Publications","issue":"4","license":[{"start":{"date-parts":[[1987,7,1]],"date-time":"1987-07-01T00:00:00Z","timestamp":552096000000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/journals.sagepub.com\/page\/policies\/text-and-data-mining-license"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Vet Pathol"],"published-print":{"date-parts":[[1987,7]]},"abstract":"<jats:p>\n                    Chronic corneal edema occurred in 53% of 116 ranch mink (\n                    <jats:italic toggle=\"yes\">Mustela vison<\/jats:italic>\n                    ) 8 to 11 years old. Most were royal pastel females, the main group at risk. Bilateral in 46 of 66 affected mink studied, the edema evolved over a month or so until the cornea became opaque, diffusely pale blue-gray or white, and greatly thickened. The swollen cornea did not become ulcerated, pigmented, or vascularized, even after it had been severely edematous for a year or two. The edema supervened as a consequence of spontaneous deterioration of the corneal endothelium. Attenuation and loss of the endothelial monolayer were the most common light microscopic changes. Other changes included discrete excrescences (guttata) along the posterior surface of the thickened Descemet's membrane and a subendothelial fibrillar or fibrocellular layer (posterior collagenous layer) often apposed to the excrescences. Likened to the primary endothelial dystrophies of man and the dog, this endothelial disorder of mink is regarded as an abiotrophic degeneration with its own distinguishing features in this species.\n                  <\/jats:p>","DOI":"10.1177\/030098588702400406","type":"journal-article","created":{"date-parts":[[2017,1,15]],"date-time":"2017-01-15T12:36:14Z","timestamp":1484483774000},"page":"323-329","source":"Crossref","is-referenced-by-count":6,"title":["Chronic Corneal Edema in Aged Ranch Mink"],"prefix":"10.1177","volume":"24","author":[{"given":"W. J.","family":"Hadlow","sequence":"first","affiliation":[{"name":"U.S. Department of Health and Human Services, Public Health Service, National Institutes of Health, National Institute of Allergy and Infectious Diseases, Laboratory of Pathobiology, Rocky Mountain Laboratories, Hamilton, MT"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"179","published-online":{"date-parts":[[1987,7]]},"reference":[{"key":"e_1_2_2_2_1","doi-asserted-by":"publisher","DOI":"10.2460\/ajvr.1981.42.04.590"},{"key":"e_1_2_2_3_1","doi-asserted-by":"publisher","DOI":"10.1001\/archopht.1982.01030040932013"},{"key":"e_1_2_2_4_1","doi-asserted-by":"publisher","DOI":"10.1016\/0002-9394(72)91200-7"},{"key":"e_1_2_2_5_1","doi-asserted-by":"publisher","DOI":"10.1016\/0531-5565(82)90023-7"},{"key":"e_1_2_2_6_1","first-page":"151","volume-title":"Spontaneous Animal Models of Human Disease","author":"Gelatt KN","year":"1979","unstructured":"Gelatt KN, Peiffer RLJr, Williams LW: Corneal dystrophies and degenerations. In: Spontaneous Animal Models of Human Disease, ed. Andrews EJ, Ward BC, Altman NH, vol. 1, pp. 151\u2013155. Academic Press, New York, 1979"},{"key":"e_1_2_2_7_1","first-page":"561","article-title":"Histopathology of corneal edema.","volume":"8","author":"Goldman JN","year":"1968","unstructured":"Goldman JN, Kuwabara T: Histopathology of corneal edema. Int Ophthalmol Clin 8: 561\u2013579, 1968","journal-title":"Int Ophthalmol Clin"},{"key":"e_1_2_2_8_1","first-page":"267","article-title":"Decrease in canine corneal endothelial cell density and increase in corneal thickness as functions of age.","volume":"22","author":"Gwin RM","year":"1982","unstructured":"Gwin RM, Lerner I, Warren JK, Gum G: Decrease in canine corneal endothelial cell density and increase in corneal thickness as functions of age. 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