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PCM in CLD is known to be associated with life\u2010threatening complications. In the general approach to these patients, the initial and most important step for the clinician is to recognize the extent of malnutrition. Most patients tolerate a normal amount of dietary protein without developing hepatic encephalopathy (HE). Oral branched\u2010chain amino acids (BCAAs) have a limited role in HE. Patients who exhibit dietary protein intolerance originally were thought to be best treated with BCAA formulations. Mixed evidence has been reported in multiple studies. In keeping with other reports, this article shows that in animal protein\u2013intolerant patients, even those with advanced cirrhosis, vegetable protein\u2013based diets are well tolerated. Another approach to management of apparent dietary intolerance is to optimize HE treatment with available medications. This article reviews the causes of HE, minimal HE, and PCM; examines nutrition requirements and assessment; and discusses treatment options for malnutrition in HE.<\/jats:p>","DOI":"10.1177\/0884533610368712","type":"journal-article","created":{"date-parts":[[2010,6,25]],"date-time":"2010-06-25T15:18:51Z","timestamp":1277479131000},"page":"257-264","source":"Crossref","is-referenced-by-count":38,"title":["Nutrition in Hepatic Encephalopathy"],"prefix":"10.1002","volume":"25","author":[{"given":"Rajagopal","family":"Chadalavada","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Raja Shekhar 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