{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,10,16]],"date-time":"2026-10-16T23:17:33Z","timestamp":1792192653442,"version":"4.4.0"},"reference-count":16,"publisher":"SAGE Publications","issue":"6","license":[{"start":{"date-parts":[[1986,12,1]],"date-time":"1986-12-01T00:00:00Z","timestamp":533779200000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/journals.sagepub.com\/page\/policies\/text-and-data-mining-license"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Human Toxicology"],"published-print":{"date-parts":[[1986,12]]},"abstract":"<jats:p>1 Blood concentrations of lactate, ketone bodies and non-esterified (free) fatty acids were measured in 45 adult patients on admission to the poisoning treatment centre, with salicylate and mixed salicylate-sedative\/ethanol poisoning.<\/jats:p>\n                  <jats:p>2 Nine patients had blood lactate concentrations above 2 mmol\/l and six patients had ketone-body concentrations greater than 1 mmol\/l.<\/jats:p>\n                  <jats:p>3 Eight of the nine patients with hyperlactacidaemia had mixed salicylate\/ethanol or sedative poisoning, whereas four of the six patients with hyperketonaemia had pure salicylate poisoning.<\/jats:p>\n                  <jats:p>4 No direct correlation was found between the presence of these metabolic disturbances and the severity of poisoning.<\/jats:p>\n                  <jats:p>5 Since only one patient was found with organic acid concentration in the blood exceeding 5 mmol\/l, it is concluded that concomitant endogenous acidosis rarely contributes to the acid-base disturbance seen in salicylate poisoning in adults.<\/jats:p>","DOI":"10.1177\/096032718600500604","type":"journal-article","created":{"date-parts":[[2008,12,24]],"date-time":"2008-12-24T01:08:04Z","timestamp":1230080884000},"page":"363-366","source":"Crossref","is-referenced-by-count":21,"title":["Blood Lactate and Ketone Body Concentrations in Salicylate Intoxication"],"prefix":"10.1177","volume":"5","author":[{"given":"P.D.","family":"Bartels","sequence":"first","affiliation":[{"name":"Poisoning Treatment Centre, Departments of Medicine C and Clinical Chemistry, Bispebjerg Hospital, DK 2400, Copenhagen N.V., Denmark"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"H.","family":"Lund-Jacobsen","sequence":"additional","affiliation":[{"name":"Poisoning Treatment Centre, Departments of Medicine C and Clinical Chemistry, Bispebjerg Hospital, DK 2400, Copenhagen N.V., Denmark"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"179","published-online":{"date-parts":[[1986,12,1]]},"reference":[{"key":"atypb1","first-page":"2833","volume":"145","author":"Bj\u00e6ldager, P.A.L.","year":"1983","journal-title":"Ugeskr. 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