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The discovery of autoantibodies in some of these patients and the introduction of molecular biology have resulted in a new understanding of the pathological mechanisms of many of the bullous lesions. In this article, updated topics of the immune-mediated bullous lesions which involve oral mucosa and skin are reviewed. Pemphigus antigens, which are desmosomal-associated proteins and belong to the cadherin superfamily of cell adhesion proteins, have been isolated, and their genes have been cloned. The antigens which react with autoantibodies from patients with bullous pemphigoid, cicatricial pemphigoid, acquired epidermolysis bullosa, and linear IgA disease are all proteins of the hemidesmosome basement membrane complex. Interestingly, most of the antigens also appear to be the target for mutations seen in patients with the inherited type of epidermolysis bullosa in which bullous lesions are a prominent clinical feature. <\/jats:p>","DOI":"10.1177\/10454411980090020201","type":"journal-article","created":{"date-parts":[[2009,1,9]],"date-time":"2009-01-09T00:40:32Z","timestamp":1231461632000},"page":"162-178","source":"Crossref","is-referenced-by-count":27,"title":["Molecular Biological Aspects of Acquired Bullous Diseases"],"prefix":"10.1177","volume":"9","author":[{"given":"E.","family":"Dabelsteen","sequence":"first","affiliation":[{"name":"Department of Oral Diagnostics, School of Dentistry, Faculty of Health Sciences, University of Copenhagen, N\u00f8rre All\u00e9 20, DK-2200 Copenhagen N, Denmark"}],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"179","published-online":{"date-parts":[[1998,4,1]]},"reference":[{"key":"atypb1","unstructured":"Alberts B., Bray D., Lewis J., Raff M., Roberts K., Watson JD (1994). 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