{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,9,8]],"date-time":"2026-09-08T00:07:33Z","timestamp":1788826053551,"version":"build-2803163510"},"reference-count":14,"publisher":"SAGE Publications","issue":"5","license":[{"start":{"date-parts":[[2026,3,21]],"date-time":"2026-03-21T00:00:00Z","timestamp":1774051200000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/journals.sagepub.com\/page\/policies\/text-and-data-mining-license"}],"content-domain":{"domain":["journals.sagepub.com"],"crossmark-restriction":true},"short-container-title":["J Vasc Access"],"published-print":{"date-parts":[[2026,9]]},"abstract":"<jats:sec>\n                    <jats:title>Background:<\/jats:title>\n                    <jats:p>Autosomal dominant polycystic kidney disease (ADPKD) is associated with distinct vascular remodeling; however, its impact on arteriovenous fistula (AVF) outcomes in hemodialysis patients remains unclear. This study aimed to compare AVF patency and survival between patients with and without ADPKD.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Methods:<\/jats:title>\n                    <jats:p>\n                      We conducted a retrospective observational cohort study including 38 patients with ADPKD and 38 age-matched non-ADPKD patients who underwent first-time AVF creation between 2013 and 2021. Patients were followed for 24\u2009months. Primary failure, primary patency, and assisted primary patency were assessed. Statistical analyses included Student\u2019s\n                      <jats:italic toggle=\"yes\">t<\/jats:italic>\n                      -test, chi-square test, and Kaplan\u2013Meier survival analysis.\n                    <\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Results:<\/jats:title>\n                    <jats:p>\n                      Compared with non-ADPKD patients, those with ADPKD had a significantly lower prevalence of diabetes mellitus (7.9% vs 52.6%,\n                      <jats:italic toggle=\"yes\">p<\/jats:italic>\n                      \u2009&lt;\u20090.001), ischemic heart disease (2.6% vs 31.6%,\n                      <jats:italic toggle=\"yes\">p<\/jats:italic>\n                      \u2009&lt;\u20090.001), and peripheral artery disease (5.3% vs 23.7%,\n                      <jats:italic toggle=\"yes\">p<\/jats:italic>\n                      \u2009=\u20090.022). Distal AVF creation was more frequent in the ADPKD group (73.7% vs 57.9%,\n                      <jats:italic toggle=\"yes\">p<\/jats:italic>\n                      \u2009=\u20090.147). At 24\u2009months, primary patency was 63.2% in the ADPKD group and 60.5% in the non-ADPKD group (\n                      <jats:italic toggle=\"yes\">p<\/jats:italic>\n                      \u2009=\u20090.81), while assisted primary patency was 84.2% and 73.7%, respectively (\n                      <jats:italic toggle=\"yes\">p<\/jats:italic>\n                      \u2009=\u20090.26). Kaplan\u2013Meier analysis showed no significant difference in AVF survival between groups (\n                      <jats:italic toggle=\"yes\">p<\/jats:italic>\n                      \u2009=\u20090.22).\n                    <\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Conclusions:<\/jats:title>\n                    <jats:p>Despite a lower burden of traditional atherosclerotic risk factors, patients with ADPKD exhibited AVF patency and survival comparable to those without ADPKD. These findings suggest that disease-specific vascular remodeling, rather than atherosclerosis alone, plays a key role in AVF outcomes. Larger prospective studies are warranted to further elucidate the mechanisms influencing vascular access performance in ADPKD.<\/jats:p>\n                  <\/jats:sec>","DOI":"10.1177\/11297298261428519","type":"journal-article","created":{"date-parts":[[2026,3,21]],"date-time":"2026-03-21T10:45:23Z","timestamp":1774089923000},"page":"2049-2054","update-policy":"https:\/\/doi.org\/10.1177\/sage-journals-update-policy","source":"Crossref","is-referenced-by-count":0,"title":["Comparison of arteriovenous fistula outcomes in patients with and without autosomal dominant polycystic kidney disease: A retrospective study"],"prefix":"10.1177","volume":"27","author":[{"ORCID":"https:\/\/orcid.org\/0009-0008-7999-5691","authenticated-orcid":false,"given":"Joana Pereira","family":"Dias","sequence":"first","affiliation":[{"name":"Nephrology Department, Unidade Local de Sa\u00fade de Vila Nova de Gaia Espinho, Vila Nova de Gaia, Portugal"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Maria Alexandra","family":"Martingo","sequence":"additional","affiliation":[{"name":"Nephrology Department, Unidade Local de Sa\u00fade de Vila Nova de Gaia Espinho, Vila Nova de Gaia, Portugal"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Maria Beatriz","family":"Bessa","sequence":"additional","affiliation":[{"name":"Nephrology Department, Unidade Local de Sa\u00fade de Vila Nova de Gaia Espinho, Vila Nova de Gaia, Portugal"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Patricia","family":"Fonseca","sequence":"additional","affiliation":[{"name":"Nephrology Department, Unidade Local de Sa\u00fade de Vila Nova de Gaia Espinho, Vila Nova de Gaia, Portugal"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Susana","family":"Pereira","sequence":"additional","affiliation":[{"name":"Nephrology Department, Unidade Local de Sa\u00fade de Vila Nova de Gaia Espinho, Vila Nova de Gaia, Portugal"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Ana","family":"Ventura","sequence":"additional","affiliation":[{"name":"Nephrology Department, Unidade Local de Sa\u00fade de Vila Nova de Gaia Espinho, Vila Nova de Gaia, Portugal"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Clara","family":"Almeida","sequence":"additional","affiliation":[{"name":"Nephrology Department, Unidade Local de Sa\u00fade de Vila Nova de Gaia Espinho, Vila Nova de Gaia, Portugal"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"179","published-online":{"date-parts":[[2026,3,21]]},"reference":[{"key":"e_1_3_2_2_2","doi-asserted-by":"publisher","DOI":"10.1038\/srep12816"},{"key":"e_1_3_2_3_2","doi-asserted-by":"publisher","DOI":"10.34067\/KID.0000000000000453"},{"key":"e_1_3_2_4_2","doi-asserted-by":"publisher","DOI":"10.2215\/CJN.05850518"},{"key":"e_1_3_2_5_2","doi-asserted-by":"publisher","DOI":"10.2215\/CJN.09441209"},{"key":"e_1_3_2_6_2","doi-asserted-by":"publisher","DOI":"10.1038\/s41598-020-58441-5"},{"key":"e_1_3_2_7_2","doi-asserted-by":"publisher","DOI":"10.1053\/j.ajkd.2019.12.001"},{"key":"e_1_3_2_8_2","doi-asserted-by":"publisher","DOI":"10.2215\/CJN.11531116"},{"key":"e_1_3_2_9_2","doi-asserted-by":"publisher","DOI":"10.2215\/CJN.13321216"},{"key":"e_1_3_2_10_2","doi-asserted-by":"publisher","DOI":"10.1111\/ijcp.13721"},{"key":"e_1_3_2_11_2","doi-asserted-by":"publisher","DOI":"10.1016\/j.semnephrol.2019.10.009"},{"key":"e_1_3_2_12_2","doi-asserted-by":"publisher","DOI":"10.1186\/s12882-021-02313-1"},{"key":"e_1_3_2_13_2","doi-asserted-by":"crossref","unstructured":"Gerald A Beathard MP. www.uptodate.com http:\/\/www.uptodate.com (n.d.). (accessed 4 December 2025).","DOI":"10.1097\/01.COT.0000000000000202"},{"issue":"4","key":"e_1_3_2_14_2","article-title":"The impact of chronic kidney disease on arteriovenous fistula\u2009remodeling: studies in a murine model of autosomal dominant polycystic kidney disease","volume":"329","author":"Laboyrie SL","year":"2025","unstructured":"Laboyrie SL, Peters DJM, Bijkerk R, et al. The impact of chronic kidney disease on arteriovenous fistula\u2009remodeling: studies in a murine model of autosomal dominant polycystic kidney disease. Physiol Renal\u2009Physiol 2025; 329(4): F453\u2013F464.","journal-title":"Physiol Renal\u2009Physiol"},{"key":"e_1_3_2_15_2","doi-asserted-by":"publisher","DOI":"10.1016\/j.jvs.2020.02.035"}],"container-title":["The Journal of Vascular Access"],"original-title":[],"language":"en","link":[{"URL":"https:\/\/journals.sagepub.com\/doi\/pdf\/10.1177\/11297298261428519","content-type":"application\/pdf","content-version":"vor","intended-application":"text-mining"},{"URL":"https:\/\/journals.sagepub.com\/doi\/full-xml\/10.1177\/11297298261428519","content-type":"application\/xml","content-version":"vor","intended-application":"text-mining"},{"URL":"https:\/\/journals.sagepub.com\/doi\/pdf\/10.1177\/11297298261428519","content-type":"unspecified","content-version":"vor","intended-application":"similarity-checking"}],"deposited":{"date-parts":[[2026,9,7]],"date-time":"2026-09-07T23:18:55Z","timestamp":1788823135000},"score":1,"resource":{"primary":{"URL":"https:\/\/journals.sagepub.com\/doi\/10.1177\/11297298261428519"}},"subtitle":[],"short-title":[],"issued":{"date-parts":[[2026,3,21]]},"references-count":14,"journal-issue":{"issue":"5","published-print":{"date-parts":[[2026,9]]}},"alternative-id":["10.1177\/11297298261428519"],"URL":"https:\/\/doi.org\/10.1177\/11297298261428519","relation":{},"ISSN":["1129-7298","1724-6032"],"issn-type":[{"value":"1129-7298","type":"print"},{"value":"1724-6032","type":"electronic"}],"subject":[],"published":{"date-parts":[[2026,3,21]]}}}