{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,5,2]],"date-time":"2026-05-02T12:38:33Z","timestamp":1777725513122,"version":"3.51.4"},"reference-count":20,"publisher":"SAGE Publications","issue":"5","license":[{"start":{"date-parts":[[2000,10,1]],"date-time":"2000-10-01T00:00:00Z","timestamp":970358400000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/journals.sagepub.com\/page\/policies\/text-and-data-mining-license"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["J Int Med Res"],"published-print":{"date-parts":[[2000,10]]},"abstract":"<jats:p>Breast-feeding helps to protect against coeliac disease because of the presence of antigliadin-IgA antibodies (AGA-IgA) in breast milk. The aim of this study was to assess the concentrations of AGA-IgA in breast milk during lactation, and whether these concentrations vary with the socioeconomic status of the women. Samples of serum for determination of IgA albumin and AGA-IgA, and samples of breast-milk for AGA-IgA were collected from 105 healthy mothers (aged 17\u201336 years). Women were divided into two groups: group 1 were of low and group 2 were of high socioeconomic status. No differences were observed between the study parameters in the two groups. Serum AGA-IgA in both groups was, however, significantly lower than that in colostrum. AGA-IgA concentrations in both groups gradually decreased during 45 days of lactation; the difference between colostrum and the samples taken at days 10 and 30\u201345 of lactation was significant. The encouragement of sufficient and long-term (e.g. 4\u20136 months) breast-feeding seems likely to be beneficial in preventing coeliac disease.<\/jats:p>","DOI":"10.1177\/147323000002800506","type":"journal-article","created":{"date-parts":[[2013,2,27]],"date-time":"2013-02-27T14:22:28Z","timestamp":1361974948000},"page":"234-240","source":"Crossref","is-referenced-by-count":6,"title":["Gliadin-Specific IgA Antibodies in Breast Milk"],"prefix":"10.1177","volume":"28","author":[{"given":"T","family":"\u00d6zkan","sequence":"first","affiliation":[{"name":"Department of Paediatric Gastroenterology, University of Uludag, Medical School, G\u00f6r\u00fcUkle-Bursa, Turkey"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"T","family":"\u00d6zeke","sequence":"additional","affiliation":[{"name":"Department of Paediatric Gastroenterology, University of Uludag, Medical School, G\u00f6r\u00fcUkle-Bursa, Turkey"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"A","family":"Meral","sequence":"additional","affiliation":[{"name":"Department of Paediatrics, University of Uludag, Medical School, G\u00f6r\u00fckle-Bursa, Turkey"}],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"179","published-online":{"date-parts":[[2000,10,1]]},"reference":[{"key":"bibr1-147323000002800506","unstructured":"Walker-Smith JA: Celiac disease. 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