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Oxidative stress extends beyond merely mediating early tissue injury; it acts as the central regulatory axis orchestrating sequential pathological transitions, encompassing epithelial damage, immune activation, chronic ischemia, and fibrotic remodeling.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Recent Advances:<\/jats:title>\n                    <jats:p>RT-induced reactive oxygen species (ROS) burst provokes lipid peroxidation, mitochondrial dysfunction, programmed cell death, and epithelial barrier disruption. Subsequently, damage-associated molecular pattern release and microbial translocation propel immune-inflammatory amplification. Sustained ROS generation and ensuing inflammation further compromise the vascular endothelium, precipitating microcirculatory dysfunction and chronic hypoxia. Concurrently, barrier disruption and microbial dysbiosis form a feedback loop shifting sustained regenerative responses toward maladaptive repair, ultimately culminating in irreversible tissue remodeling.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Critical Issues:<\/jats:title>\n                    <jats:p>Pathogenically, RE is not simply a consequence of ROS overload, but an ROS-driven continuum of tissue state transitions resulting in regenerative failure. Furthermore, a \u201credox paradox\u201d emerges wherein ROS concurrently drive normal intestinal injury and mediate RT\u2019s tumoricidal efficacy. Currently, regenerative failure and this redox paradox remain inadequately integrated into clinical paradigms for patient stratification, interventional timing, and efficacy evaluation.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Future Directions:<\/jats:title>\n                    <jats:p>\n                      Future endeavors must prioritize precision redox therapeutics characterized by tissue specificity, temporal regulation, and dose-dependent modulation. Leveraging targeted delivery, phased interventions, and biomarkers will reconcile the conflicting demands of normal tissue radioprotection and tumor control, propelling the paradigm shift from broad-spectrum antioxidants toward precision redox medicine.\n                      <jats:italic toggle=\"yes\">Antioxid. Redox Signal.<\/jats:italic>\n                      45, 596\u2013617.\n                    <\/jats:p>\n                  <\/jats:sec>","DOI":"10.1177\/15230864261481021","type":"journal-article","created":{"date-parts":[[2026,8,20]],"date-time":"2026-08-20T02:32:06Z","timestamp":1787193126000},"page":"596-617","update-policy":"https:\/\/doi.org\/10.1177\/sage-journals-update-policy","source":"Crossref","is-referenced-by-count":0,"title":["Unraveling Radiation Enteritis: Oxidative Stress as a Central Driver and Therapeutic Target"],"prefix":"10.1177","volume":"45","author":[{"ORCID":"https:\/\/orcid.org\/0009-0001-6021-2625","authenticated-orcid":false,"given":"Pengfei","family":"Zhong","sequence":"first","affiliation":[{"name":"The First School of Clinical Medicine, Gansu University of Chinese Medicine (Gansu Provincial Hospital), Lanzhou, China."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Zhiyuan","family":"Li","sequence":"additional","affiliation":[{"name":"The First School of Clinical Medicine, Gansu University of Chinese Medicine (Gansu Provincial Hospital), Lanzhou, China."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Zhenjun","family":"Pu","sequence":"additional","affiliation":[{"name":"The First School of Clinical Medicine, Gansu University of Chinese Medicine (Gansu Provincial Hospital), Lanzhou, China."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Hongyu","family":"Nian","sequence":"additional","affiliation":[{"name":"The First School of Clinical Medicine, Gansu University of Chinese Medicine (Gansu Provincial Hospital), Lanzhou, China."}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Junliang","family":"Li","sequence":"additional","affiliation":[{"name":"The First School of Clinical Medicine, Gansu University of Chinese Medicine (Gansu Provincial Hospital), Lanzhou, China."},{"name":"Department of General Surgery, Gansu Provincial Hospital, Lanzhou, China."},{"name":"Institute of Modern Physics, Chinese Academy of Sciences, Lanzhou, China."}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"179","published-online":{"date-parts":[[2026,8,19]]},"reference":[{"key":"e_1_3_2_2_1","doi-asserted-by":"publisher","DOI":"10.1016\/j.ijrobp.2023.09.021"},{"key":"e_1_3_2_3_1","doi-asserted-by":"publisher","DOI":"10.1016\/j.lfs.2024.122921"},{"key":"e_1_3_2_4_1","unstructured":"American Journal of Physiology-Gastrointestinal and Liver Physiology. 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