{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,5,4]],"date-time":"2026-05-04T06:39:47Z","timestamp":1777876787264,"version":"3.51.4"},"reference-count":33,"publisher":"SAGE Publications","issue":"1","license":[{"start":{"date-parts":[[2015,12,11]],"date-time":"2015-12-11T00:00:00Z","timestamp":1449792000000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/journals.sagepub.com\/page\/policies\/text-and-data-mining-license"}],"content-domain":{"domain":["journals.sagepub.com"],"crossmark-restriction":true},"short-container-title":["Neuroradiol J"],"published-print":{"date-parts":[[2016,2]]},"abstract":"<jats:sec>\n                    <jats:title>Objective<\/jats:title>\n                    <jats:p>To evaluate the role of advanced magnetic resonance (MR) sequences (fast imaging employing steady-state acquisition (FIESTA), T2 star-weighted angiography (SWAN) and spoiled gradient recalled echo (SPGR)) in patients with single small enhancing computed tomography lesions and scolex demonstration in typical and atypical parenchymal neurocysticercosis.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Methods<\/jats:title>\n                    <jats:p>In this study, 59 patients of new-onset seizures with single small enhancing computed tomography lesions of the brain were included. Along with routine MR sequences, advanced MR sequences, like SWAN, FIESTA, and pre and post-contrast SPGR, were performed. Follow-up MR studies focussing on the morphology of the lesions and demonstration of scolex were performed 6 monthly for 3 years.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Results<\/jats:title>\n                    <jats:p>The majority of patients (62.7%) were men with partial seizure as the most common manifestation. On SPGR, contrast lesions were identified as either \u2018typical\u2019 (42, 71.2%) or \u2018atypical\u2019 (17, 28.8%). In the typical lesion group, SWAN and FIESTA sequences detected scolex in 30 (71.4%) and 32 (76.2%), respectively. The combination of SPGR-contrast, FIESTA and SWAN sequences detected scolex in 35 (83.3%) patients compared to 19 (45.2%) by routine sequences ( P\u2009&lt;\u20090.001). In the atypical lesion group, SWAN and FIESTA sequences detected scolex in 15 (88.2%) and 16 (94.1%) patients, respectively. The combination of SPGR-contrast, FIESTA and SWAN sequences detected scolex in 16 (94.1%) patients compared to 10 (58.8%) by routine sequences ( P\u2009&lt;\u20090.001). Follow-up showed greater resolution with lesser calcification in the typical group compared to the atypical group.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Conclusion<\/jats:title>\n                    <jats:p>This study provides an insight into the natural course of typical and atypical solitary cysticercus granuloma lesions, and the utility of SPGR-contrast, FIESTA and SWAN MR sequences in scolex demonstration and identification of atypical lesions.<\/jats:p>\n                  <\/jats:sec>","DOI":"10.1177\/1971400915620437","type":"journal-article","created":{"date-parts":[[2015,12,11]],"date-time":"2015-12-11T21:49:55Z","timestamp":1449870595000},"page":"19-29","update-policy":"https:\/\/doi.org\/10.1177\/sage-journals-update-policy","source":"Crossref","is-referenced-by-count":7,"title":["Natural course of typical and atypical parenchymal solitary cysticercus granuloma of the brain: a 3-year prospective clinico-radiological study"],"prefix":"10.1177","volume":"29","author":[{"given":"Neeraj","family":"Kumar","sequence":"first","affiliation":[{"name":"Department of Neurology, King George Medical University, India"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Ravindra Kumar","family":"Garg","sequence":"additional","affiliation":[{"name":"Department of Neurology, King George Medical University, India"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Hardeep Singh","family":"Malhotra","sequence":"additional","affiliation":[{"name":"Department of Neurology, King George Medical University, India"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Rakesh Kumar","family":"Gupta","sequence":"additional","affiliation":[{"name":"MR Section, Department of Radiodiagnosis, Sanjay Gandhi Postgraduate Institute of Medical Sciences, India"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Rajesh","family":"Verma","sequence":"additional","affiliation":[{"name":"Department of Neurology, King George Medical University, India"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Praveen Kumar","family":"Sharma","sequence":"additional","affiliation":[{"name":"Department of Neurology, King George Medical University, India"}],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"179","published-online":{"date-parts":[[2015,12,11]]},"reference":[{"key":"bibr1-1971400915620437","doi-asserted-by":"publisher","DOI":"10.1046\/j.1365-3156.2000.00541.x"},{"key":"bibr2-1971400915620437","doi-asserted-by":"publisher","DOI":"10.1016\/S0001-706X(03)00055-X"},{"key":"bibr3-1971400915620437","doi-asserted-by":"publisher","DOI":"10.3171\/foc.2002.12.6.5"},{"key":"bibr4-1971400915620437","doi-asserted-by":"publisher","DOI":"10.1586\/eri.12.35"},{"key":"bibr5-1971400915620437","unstructured":"Rajshekhar V and Chandy MJ (eds). 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