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Five hundred and six participants with psychosis-like experiences (PLEs) and 328 control subjects performed behavioral tasks measuring these mechanisms in addition to symptom assessments. Participants\u2019 childhood-area social fragmentation and childhood opportunities were also extracted. The association between environmental, computational, and clinical measures was assessed, as were mediation pathways. Higher social fragmentation was associated with deficits in RL,\n                    <jats:italic toggle=\"yes\">r<\/jats:italic>\n                    (410)\u2009=\u2009.19,\n                    <jats:italic toggle=\"yes\">p<\/jats:italic>\n                    \u2009&lt;\u2009.001, and lower opportunities were associated with overweighting perceptual priors,\n                    <jats:italic toggle=\"yes\">r<\/jats:italic>\n                    (290)\u2009=\u2009\u2212.16,\n                    <jats:italic toggle=\"yes\">p<\/jats:italic>\n                    \u2009=\u2009.02, and issues in RL,\n                    <jats:italic toggle=\"yes\">r<\/jats:italic>\n                    (307)\u2009=\u2009\u2212.14,\n                    <jats:italic toggle=\"yes\">p<\/jats:italic>\n                    \u2009=\u2009.02, for PLE participants. Fewer available childhood opportunities were also related to greater positive symptom severity,\n                    <jats:italic toggle=\"yes\">r<\/jats:italic>\n                    (354)\u2009=\u2009\u2212.14,\n                    <jats:italic toggle=\"yes\">p<\/jats:italic>\n                    \u2009=\u2009.03. We found a potential pathway connecting available childhood opportunities to positive symptoms through the mediating effect of overweighting perceptual priors (average causal mediation effect\u2009=\u2009\u22124.91,\n                    <jats:italic toggle=\"yes\">p<\/jats:italic>\n                    \u2009=\u2009.04). Environmental characteristics could shape symptom severity through computational mechanisms.\n                  <\/jats:p>","DOI":"10.1177\/21677026261488357","type":"journal-article","created":{"date-parts":[[2026,10,13]],"date-time":"2026-10-13T06:17:53Z","timestamp":1791872273000},"update-policy":"https:\/\/doi.org\/10.1177\/sage-journals-update-policy","source":"Crossref","is-referenced-by-count":0,"title":["Environmental Exposures and Computational Mechanisms in Psychosis Risk: Insights From a Multisite Cohort"],"prefix":"10.1177","author":[{"ORCID":"https:\/\/orcid.org\/0000-0002-7648-9215","authenticated-orcid":false,"given":"Luz Maria","family":"Alliende","sequence":"first","affiliation":[{"name":"Department of Psychology, Northwestern University"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"ORCID":"https:\/\/orcid.org\/0000-0002-1003-0196","authenticated-orcid":false,"given":"Victor","family":"Pokorny","sequence":"additional","affiliation":[{"name":"Department of Psychology, Northwestern University"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"ORCID":"https:\/\/orcid.org\/0000-0001-6994-8770","authenticated-orcid":false,"given":"Benson","family":"Ku","sequence":"additional","affiliation":[{"name":"Department of Psychiatry and Behavioral Sciences, Emory University"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"ORCID":"https:\/\/orcid.org\/0009-0008-3340-9255","authenticated-orcid":false,"given":"Natalie","family":"Larson","sequence":"additional","affiliation":[{"name":"Department of Psychology, Northwestern University"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"ORCID":"https:\/\/orcid.org\/0000-0002-7218-5169","authenticated-orcid":false,"given":"Gregory P.","family":"Strauss","sequence":"additional","affiliation":[{"name":"Departments of Psychology and Neuroscience, University of Georgia"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"ORCID":"https:\/\/orcid.org\/0009-0006-8852-0829","authenticated-orcid":false,"given":"L. 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