{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2022,4,4]],"date-time":"2022-04-04T15:46:14Z","timestamp":1649087174742},"reference-count":7,"publisher":"Springer Science and Business Media LLC","issue":"1","license":[{"start":{"date-parts":[[2010,7,30]],"date-time":"2010-07-30T00:00:00Z","timestamp":1280448000000},"content-version":"unspecified","delay-in-days":0,"URL":"http:\/\/www.springer.com\/tdm"}],"content-domain":{"domain":["link.springer.com"],"crossmark-restriction":false},"short-container-title":["J Med Case Reports"],"published-print":{"date-parts":[[2010,12]]},"abstract":"<jats:title>Abstract<\/jats:title>\n          <jats:sec>\n            <jats:title>Introduction<\/jats:title>\n            <jats:p>To the best of our knowledge, this is the first report in the literature of development of neurogenic diabetes insipidus in a patient with subacute liver failure.<\/jats:p>\n          <\/jats:sec>\n          <jats:sec>\n            <jats:title>Case presentation<\/jats:title>\n            <jats:p>A 25-year-old man presented with subacute liver failure. While awaiting a liver transplant, the patient developed cerebral edema, which resulted in neurogenic diabetes insipidus secondary to cerebral edema. The patient died before the liver transplantation could be carried out.<\/jats:p>\n          <\/jats:sec>\n          <jats:sec>\n            <jats:title>Conclusion<\/jats:title>\n            <jats:p>Neurogenic diabetes insipidus is well recognized in the neurosurgical population as a consequence of cerebral edema and increased intracranial pressure, both of which occur commonly in patients with subacute liver failure.<\/jats:p>\n          <\/jats:sec>","DOI":"10.1186\/1752-1947-4-232","type":"journal-article","created":{"date-parts":[[2010,7,30]],"date-time":"2010-07-30T18:59:21Z","timestamp":1280516361000},"update-policy":"http:\/\/dx.doi.org\/10.1007\/springer_crossmark_policy","source":"Crossref","is-referenced-by-count":1,"title":["Neurogenic diabetes insipidus presenting in a patient with subacute liver failure: a case report"],"prefix":"10.1186","volume":"4","author":[{"given":"Manu","family":"Shankar Hari","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Anthony K","family":"Parsons","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Andy K","family":"Burroughs","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Steve","family":"Shaw","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"James","family":"O'Beirne","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Banwari","family":"Agarwal","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"297","published-online":{"date-parts":[[2010,7,30]]},"reference":[{"key":"1189_CR1","doi-asserted-by":"publisher","first-page":"5987","DOI":"10.1210\/jc.2004-1058","volume":"89","author":"A Agha","year":"2004","unstructured":"Agha A, Thornton E, O'Kelly P, Tormey W, Phillips W, Thompson CJ: Posterior pituitary dysfunction after traumatic brain injury. J Clin Endocrinol Metab. 2004, 89: 5987-5992. 10.1210\/jc.2004-1058.","journal-title":"J Clin Endocrinol Metab"},{"key":"1189_CR2","doi-asserted-by":"publisher","first-page":"124","DOI":"10.1016\/S0168-8278(05)80532-X","volume":"17","author":"A Pauwels","year":"1993","unstructured":"Pauwels A, Mostefa-Kara N, Florent C, Levy VG: Emergency liver transplantation for acute liver failure. Evaluation of London and Clichy criteria. J Hepatol. 1993, 17: 124-127. 10.1016\/S0168-8278(05)80532-X.","journal-title":"J Hepatol"},{"key":"1189_CR3","doi-asserted-by":"publisher","first-page":"145","DOI":"10.1016\/S0168-8278(96)80066-3","volume":"25","author":"FS Larsen","year":"1996","unstructured":"Larsen FS, Adel Hansen B, Pott F, Ejlersen E, Secher NH, Paulson OB, Knudsen GM: Dissociated cerebral vasoparalysis in acute liver failure. A hypothesis of gradual cerebral hyperaemia. J Hepatol. 1996, 25: 145-151. 10.1016\/S0168-8278(96)80066-3.","journal-title":"J Hepatol"},{"key":"1189_CR4","doi-asserted-by":"publisher","first-page":"80","DOI":"10.1002\/hep.1840190114","volume":"19","author":"S Aggarwal","year":"1994","unstructured":"Aggarwal S, Kramer D, Obrist W, Kang Y, Martin M, Policare R: Cerebral hemodynamic and metabolic changes in fulminant hepatic failure: a retrospective study. Hepatology. 1994, 19: 80-87. 10.1002\/hep.1840190114.","journal-title":"Hepatology"},{"key":"1189_CR5","doi-asserted-by":"publisher","first-page":"181","DOI":"10.1007\/s11011-005-7206-z","volume":"20","author":"P Ranjan","year":"2005","unstructured":"Ranjan P, Mishra AM, Kale R, Saraswat VA, Gupta RK: Cytotoxic edema is responsible for raised intracranial pressure in fulminant hepatic failure: in vivo demonstration using diffusion-weighted MRI in human subjects. Metab Brain Dis. 2005, 20: 181-192. 10.1007\/s11011-005-7206-z.","journal-title":"Metab Brain Dis"},{"key":"1189_CR6","doi-asserted-by":"publisher","first-page":"119","DOI":"10.4065\/70.2.119","volume":"70","author":"EF Wijdicks","year":"1995","unstructured":"Wijdicks EF, Plevak DJ, Ravel J, Wiesner RH: Clinical and radiologic features of cerebral edema in fulminant hepatic failure. Mayo Clin Proc. 1995, 70: 119-124. 10.4065\/70.2.119.","journal-title":"Mayo Clin Proc"},{"key":"1189_CR7","doi-asserted-by":"publisher","first-page":"1","DOI":"10.1002\/hep.1840160102","volume":"16","author":"SD Lidofsky","year":"1992","unstructured":"Lidofsky SD, Bass NM, Prager MC: Intracranial pressure monitoring and liver transplantation for fulminant hepatic failure. Hepatology. 1992, 16: 1-7. 10.1002\/hep.1840160102.","journal-title":"Hepatology"}],"container-title":["Journal of Medical Case Reports"],"original-title":[],"language":"en","link":[{"URL":"http:\/\/link.springer.com\/content\/pdf\/10.1186\/1752-1947-4-232.pdf","content-type":"application\/pdf","content-version":"vor","intended-application":"text-mining"},{"URL":"http:\/\/link.springer.com\/article\/10.1186\/1752-1947-4-232\/fulltext.html","content-type":"text\/html","content-version":"vor","intended-application":"text-mining"},{"URL":"http:\/\/link.springer.com\/content\/pdf\/10.1186\/1752-1947-4-232","content-type":"unspecified","content-version":"vor","intended-application":"similarity-checking"},{"URL":"https:\/\/link.springer.com\/content\/pdf\/10.1186\/1752-1947-4-232.pdf","content-type":"application\/pdf","content-version":"vor","intended-application":"similarity-checking"}],"deposited":{"date-parts":[[2021,9,1]],"date-time":"2021-09-01T00:48:18Z","timestamp":1630457298000},"score":1,"resource":{"primary":{"URL":"https:\/\/jmedicalcasereports.biomedcentral.com\/articles\/10.1186\/1752-1947-4-232"}},"subtitle":[],"short-title":[],"issued":{"date-parts":[[2010,7,30]]},"references-count":7,"journal-issue":{"issue":"1","published-print":{"date-parts":[[2010,12]]}},"alternative-id":["1189"],"URL":"https:\/\/doi.org\/10.1186\/1752-1947-4-232","relation":{},"ISSN":["1752-1947"],"issn-type":[{"value":"1752-1947","type":"electronic"}],"subject":[],"published":{"date-parts":[[2010,7,30]]},"assertion":[{"value":"4 June 2008","order":1,"name":"received","label":"Received","group":{"name":"ArticleHistory","label":"Article History"}},{"value":"30 July 2010","order":2,"name":"accepted","label":"Accepted","group":{"name":"ArticleHistory","label":"Article History"}},{"value":"30 July 2010","order":3,"name":"first_online","label":"First Online","group":{"name":"ArticleHistory","label":"Article History"}}],"article-number":"232"}}