{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2025,9,25]],"date-time":"2025-09-25T17:08:47Z","timestamp":1758820127458},"reference-count":14,"publisher":"Wiley","issue":"1","license":[{"start":{"date-parts":[[2006,1,12]],"date-time":"2006-01-12T00:00:00Z","timestamp":1137024000000},"content-version":"vor","delay-in-days":468,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Annals of the New York Academy of Sciences"],"published-print":{"date-parts":[[2004,10]]},"abstract":"<jats:p><jats:bold>A<jats:sc>bstract<\/jats:sc>: <\/jats:bold> Pharmacological actions of methamphetamine (METH) and phencyclidine (PCP) are different, but both of them can induce similar psychiatric disorders including abuse, intoxication, withdrawal, and psychotic symptoms like those of schizophrenia. These mental disorders are caused not only by their direct pharmacological effects, but also by secondary brain damage containing gene expression changes. In order to broadly grasp these alterations, we used serial analysis of gene expression (SAGE), a transcriptome analysis. We analyzed three cDNA libraries from cerebral cortices of saline (1 mL\/kg)\u2010, METH (4 mg\/kg)\u2010, or PCP (10 mg\/kg)\u2010treated Wistar rats (one hour after i.p. administration). The numbers of total tags were about 50,000 in each library, and approximately 18,000 kinds of tags were identified respectively. From the comparisons of three groups, we found both METH\u2010 and PCP\u2010reactive genes. Upregulated genes contained calmodulin 2, stromal cell\u2010derived factor receptor 1, brain\u2010specific angiogenesis inhibitor 1\u2010associated protein 2, ras homologue enriched in brain, basigin and thyrotropin\u2010releasing hormone receptor. Downregulated genes contained lipocalin 2, aldolase A, importin 13, fatty acid binding protein 3, and glycine receptor \u03b12 subunit. These data suggest important clues of common molecular basis in METH\u2010 and PCP\u2010related psychiatric disorders.<\/jats:p>","DOI":"10.1196\/annals.1316.007","type":"journal-article","created":{"date-parts":[[2004,11,12]],"date-time":"2004-11-12T22:34:16Z","timestamp":1100298856000},"page":"57-61","source":"Crossref","is-referenced-by-count":11,"title":["Serial Analysis of Gene Expression in Methamphetamine\u2010 and Phencyclidine\u2010Treated Rodent Cerebral Cortices: Are There Common Mechanisms?"],"prefix":"10.1111","volume":"1025","author":[{"given":"YUTA","family":"OUCHI","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"YASUHIKO","family":"KUBOTA","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"CHIHIRO","family":"ITO","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"311","published-online":{"date-parts":[[2006,1,12]]},"reference":[{"key":"e_1_2_7_2_2","doi-asserted-by":"publisher","DOI":"10.1093\/schbul\/18.1.115"},{"key":"e_1_2_7_3_2","unstructured":"Zukin S.R. 2000. Phencyclidine (or phencyclidine\u2010like)\u2010related disorders. In Kaplan & Sadock's Comprehensive Textbook of Psychiatry. B.J. Sadock & V.A. Sadock Eds.: 1063\u20101071. Lippincott Williams & Wilkins. Philadelphia."},{"key":"e_1_2_7_4_2","doi-asserted-by":"publisher","DOI":"10.1016\/S0165-0173(02)00233-3"},{"key":"e_1_2_7_5_2","unstructured":"Gur R.E. & R.C. Gur. 2000. Schizophrenia: brain structure and function.InKaplan & Sadock's Comprehensive Textbook of Psychiatry. V.A. Sadock Ed.: 1117\u20101129. Lippincott Williams & Wilkins. Philadelphia."},{"key":"e_1_2_7_6_2","doi-asserted-by":"publisher","DOI":"10.1126\/science.270.5235.484"},{"key":"e_1_2_7_7_2","unstructured":"Voet D. & J.G. Voet. 1995. Biochemistry. Wiley. 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