{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2025,10,7]],"date-time":"2025-10-07T05:14:25Z","timestamp":1759814065133,"version":"3.37.3"},"reference-count":37,"publisher":"The Endocrine Society","issue":"3","license":[{"start":{"date-parts":[[2023,10,27]],"date-time":"2023-10-27T00:00:00Z","timestamp":1698364800000},"content-version":"vor","delay-in-days":0,"URL":"https:\/\/academic.oup.com\/pages\/standard-publication-reuse-rights"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":[],"published-print":{"date-parts":[[2024,2,20]]},"abstract":"<jats:title>Abstract<\/jats:title>\n               <jats:sec>\n                  <jats:title>Context<\/jats:title>\n                  <jats:p>We recently reported that the presence of glutamic acid decarboxylase antibodies (GADA) was not associated with large-for-gestational-age infants in women with hyperglycemia in pregnancy (HIP).<\/jats:p>\n               <\/jats:sec>\n               <jats:sec>\n                  <jats:title>Objective<\/jats:title>\n                  <jats:p>We explored the association between the presence of GADA and other HIP-related adverse pregnancy outcomes.<\/jats:p>\n               <\/jats:sec>\n               <jats:sec>\n                  <jats:title>Methods<\/jats:title>\n                  <jats:p>This observational prospective study, conducted at a university hospital in a suburb of Paris, France, included 1182 consecutive women with HIP measured for GADA at HIP care initiation between 2012 and 2017. Post hoc analyses for outcomes included gestational weight gain, insulin therapy, cesarean delivery, hypertensive disorders, small-for-gestational-age infant, prematurity, and neonatal hypoglycemia.<\/jats:p>\n               <\/jats:sec>\n               <jats:sec>\n                  <jats:title>Results<\/jats:title>\n                  <jats:p>Of the 1182 women studied, 87 (7.4%) had positive (\u2265\u20051\u2005IU\/mL) GADA. Although socioeconomic, clinical, and biological characteristics were similar across women in the positive and negative GADA groups, higher fasting plasma glucose values during early HIP screening were observed in the former (5.5 \u00b1 1.5 vs 5.2 \u00b1 0.7\u2005mmol\/L respectively, P &amp;lt; .001). At HIP care initiation, fructosamine levels were higher in women with positive GADA (208 \u00b1 23 vs 200 \u00b1 18\u2005\u00b5mol\/L; P &amp;lt; .05). In the homeostatic model assessment, insulin resistance (HOMA-IR) and beta secretion (HOMA-B) rates were similar in both groups. Gestational weight gain and the rates of all adverse outcomes were similar in both groups except for cesarean delivery (18.4 and 27.3% for positive and negative GADA, respectively; adjusted odds ratio 0.49 [95% CI, 0.26-0.92], P = .026).<\/jats:p>\n               <\/jats:sec>\n               <jats:sec>\n                  <jats:title>Conclusion<\/jats:title>\n                  <jats:p>Universal measurement of GADA in women with HIP highlighted that 7.4% had positive GADA. No association was observed between GADA and HIP-related adverse pregnancy outcomes, except a lower risk of cesarean delivery.<\/jats:p>\n               <\/jats:sec>","DOI":"10.1210\/clinem\/dgad617","type":"journal-article","created":{"date-parts":[[2023,10,26]],"date-time":"2023-10-26T12:33:45Z","timestamp":1698323625000},"page":"e1117-e1124","source":"Crossref","is-referenced-by-count":3,"title":["The Prognosis of Glutamic Acid Decarboxylase Antibodies in Women With Hyperglycemia in Pregnancy"],"prefix":"10.1210","volume":"109","author":[{"ORCID":"https:\/\/orcid.org\/0000-0002-8785-3385","authenticated-orcid":false,"given":"Emmanuel","family":"Cosson","sequence":"first","affiliation":[{"name":"AP-HP, Avicenne Hospital, Paris 13 University, Sorbonne Paris Cit\u00e9, Department of Endocrinology-Diabetology-Nutrition, CRNH-IdF, CINFO , 93000 Bobigny , France"},{"name":"Equipe de Recherche en Epid\u00e9miologie Nutritionnelle (EREN), Universit\u00e9 Sorbonne Paris Nord and Universit\u00e9 Paris Cit\u00e9, INSERM, INRAE, CNAM, Center of Research in Epidemiology and StatisticS (CRESS) , 93000 Bobigny , France"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"ORCID":"https:\/\/orcid.org\/0000-0002-0816-1728","authenticated-orcid":false,"given":"Elhadji Mamadou Moussa","family":"Thioye","sequence":"additional","affiliation":[{"name":"AP-HP, Avicenne Hospital, Paris 13 University, Sorbonne Paris Cit\u00e9, Department of Endocrinology-Diabetology-Nutrition, CRNH-IdF, CINFO , 93000 Bobigny , France"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"ORCID":"https:\/\/orcid.org\/0000-0001-7358-8667","authenticated-orcid":false,"given":"Sopio","family":"Tatulashvili","sequence":"additional","affiliation":[{"name":"AP-HP, Avicenne Hospital, Paris 13 University, Sorbonne Paris Cit\u00e9, Department of Endocrinology-Diabetology-Nutrition, CRNH-IdF, CINFO , 93000 Bobigny , France"},{"name":"Equipe de Recherche en Epid\u00e9miologie Nutritionnelle (EREN), Universit\u00e9 Sorbonne Paris Nord and Universit\u00e9 Paris Cit\u00e9, INSERM, INRAE, CNAM, Center of Research in Epidemiology and StatisticS (CRESS) , 93000 Bobigny , France"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"ORCID":"https:\/\/orcid.org\/0000-0001-6303-8557","authenticated-orcid":false,"given":"Eric","family":"Vicaut","sequence":"additional","affiliation":[{"name":"AP-HP, Unit\u00e9 de Recherche Clinique St-Louis-Lariboisi\u00e8re, Universit\u00e9 Denis Diderot , 75010 Paris , France"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"ORCID":"https:\/\/orcid.org\/0000-0002-1017-1845","authenticated-orcid":false,"given":"Etienne","family":"Larger","sequence":"additional","affiliation":[{"name":"Institut Cochin, CNRS, Inserm, Universit\u00e9 de Paris , 75014 Paris , France"},{"name":"Service de Diab\u00e9tologie et Immunologie Clinique, Cochin Hospital, Assistance Publique H\u00f4pitaux de Paris, H\u00f4pitaux Universitaires de Paris Centre-Universit\u00e9 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