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Echo parameters for differential diagnosis in cardiac amyloidosis: A head-to-head comparison of deformation and nondeformation parameters. <i>Circ Cardiovasc Imaging<\/i> 2017; 10: e005588.","DOI":"10.1161\/CIRCIMAGING.116.005588"},{"key":"11","doi-asserted-by":"crossref","unstructured":"11. Phelan D, Collier P, Thavendiranathan P, Popovic ZB, Hanna M, Plana JC, et al. Relative apical sparing of longitudinal strain using two-dimensional speckle-tracking echocardiography is both sensitive and specific for the diagnosis of cardiac amyloidosis. <i>Heart<\/i> 2012; 98: 1442\u20131448.","DOI":"10.1136\/heartjnl-2012-302353"},{"key":"12","doi-asserted-by":"crossref","unstructured":"12. Lee GY, Kim HK, Choi JO, Chang SA, Oh JK, Jeon ES, et al. Visual assessment of relative apical sparing pattern is more useful than quantitative assessment for diagnosing cardiac amyloidosis in borderline or mildly increased left ventricular wall thickness. <i>Circ J<\/i> 2015; 79: 1575\u20131584.","DOI":"10.1253\/circj.CJ-14-1328"},{"key":"13","doi-asserted-by":"crossref","unstructured":"13. Wechalekar AD, Gillmore JD, Hawkins PN. Systemic amyloidosis. <i>Lancet (London, England)<\/i> 2016; 387: 2641\u20132654.","DOI":"10.1016\/S0140-6736(15)01274-X"},{"key":"14","doi-asserted-by":"crossref","unstructured":"14. Lang RM, Badano LP, Mor-Avi V, Afilalo J, Armstrong A, Ernande L, et al. Recommendations for cardiac chamber quantification by echocardiography in adults: An update from the American Society of Echocardiography and the European Association of Cardiovascular Imaging. <i>Eur Heart J Cardiovasc Imaging<\/i> 2015; 16: 233\u2013270.","DOI":"10.1093\/ehjci\/jev014"},{"key":"15","doi-asserted-by":"crossref","unstructured":"15. 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Definitions for a common standard for 2D speckle tracking echocardiography: Consensus document of the EACVI\/ASE\/Industry Task Force to standardize deformation imaging. <i>Eur Heart J Cardiovasc Imaging<\/i> 2015; 16: 1\u201311.","DOI":"10.1093\/ehjci\/jeu184"},{"key":"18","doi-asserted-by":"crossref","unstructured":"18. Perugini E, Guidalotti PL, Salvi F, Cooke RMT, Pettinato C, Riva L, et al. Noninvasive etiologic diagnosis of cardiac amyloidosis using 99 mTc-3,3-diphosphono-1,2-propanodicarboxylic acid scintigraphy. <i>J Am Coll Cardiol<\/i> 2005; 46: 1076\u20131084.","DOI":"10.1016\/j.jacc.2005.05.073"},{"key":"19","doi-asserted-by":"crossref","unstructured":"19. Damy T, Maurer MS, Rapezzi C, Plante-Bordeneuve V, Karayal ON, Mundayat R, et al. Clinical, ECG and echocardiographic clues to the diagnosis of TTR-related cardiomyopathy. <i>Open Heart<\/i> 2016; 3: e000289.","DOI":"10.1136\/openhrt-2015-000289"},{"key":"20","doi-asserted-by":"crossref","unstructured":"20. Ternacle J, Bodez D, Guellich A, Audureau E, Rappeneau S, Lim P, et al. Causes and consequences of longitudinal LV dysfunction assessed by 2D strain echocardiography in cardiac amyloidosis. <i>JACC Cardiovasc Imaging<\/i> 2016; 9: 126\u2013138.","DOI":"10.1016\/j.jcmg.2015.05.014"},{"key":"21","doi-asserted-by":"crossref","unstructured":"21. Marwick TH, Leano RL, Brown J, Sun JP, Hoffmann R, Lysyansky P, et al. Myocardial strain measurement with 2-dimensional speckle-tracking echocardiography: Definition of normal range. <i>JACC Cardiovasc Imaging<\/i> 2009; 2: 80\u201384.","DOI":"10.1016\/j.jcmg.2007.12.007"},{"key":"22","doi-asserted-by":"crossref","unstructured":"22. Hoffmann R, Altiok E, Friedman Z, Becker M, Frick M. Myocardial deformation imaging by two-dimensional speckle-tracking echocardiography in comparison to late gadolinium enhancement cardiac magnetic resonance for analysis of myocardial fibrosis in severe aortic stenosis. <i>Am J Cardiol<\/i> 2014; 114: 1083\u20131088.","DOI":"10.1016\/j.amjcard.2014.07.018"},{"key":"23","doi-asserted-by":"crossref","unstructured":"23. Weidemann F, Herrmann S, Stork S, Niemann M, Frantz S, Lange V, et al. Impact of myocardial fibrosis in patients with symptomatic severe aortic stenosis. <i>Circulation<\/i> 2009; 120: 577\u2013584.","DOI":"10.1161\/CIRCULATIONAHA.108.847772"},{"key":"24","doi-asserted-by":"crossref","unstructured":"24. Kristen AV, Schnabel PA, Winter B, Helmke BM, Longerich T, Hardt S, et al. High prevalence of amyloid in 150 surgically removed heart valves: A comparison of histological and clinical data reveals a correlation to atheroinflammatory conditions. <i>Cardiovasc Pathol<\/i> 2010; 19: 228\u2013235.","DOI":"10.1016\/j.carpath.2009.04.005"},{"key":"25","doi-asserted-by":"crossref","unstructured":"25. Scully PR, Treibel TA, Fontana M, Lloyd G, Mullen M, Pugliese F, et al. Prevalence of cardiac amyloidosis in patients referred for transcatheter aortic valve replacement. <i>J Am Coll Cardiol<\/i> 2018; 71: 463\u2013464.","DOI":"10.1016\/j.jacc.2017.11.037"},{"key":"26","doi-asserted-by":"crossref","unstructured":"26. Cavalcante JL, Rijal S, Abdelkarim I, Althouse AD, Sharbaugh MS, Fridman Y, et al. Cardiac amyloidosis is prevalent in older patients with aortic stenosis and carries worse prognosis. <i>J Cardiovasc Magn Reson<\/i> 2017; 19: 98.","DOI":"10.1186\/s12968-017-0415-x"},{"key":"27","doi-asserted-by":"crossref","unstructured":"27. 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