{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,6,8]],"date-time":"2026-06-08T23:32:08Z","timestamp":1780961528681,"version":"3.54.1"},"reference-count":7,"publisher":"SAGE Publications","issue":"4","license":[{"start":{"date-parts":[[2012,4,27]],"date-time":"2012-04-27T00:00:00Z","timestamp":1335484800000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/journals.sagepub.com\/page\/policies\/text-and-data-mining-license"}],"content-domain":{"domain":["journals.sagepub.com"],"crossmark-restriction":true},"short-container-title":["Ann Clin Biochem"],"published-print":{"date-parts":[[2012,7]]},"abstract":"<jats:sec>\n                    <jats:title>Background<\/jats:title>\n                    <jats:p>There is growing epidemiological evidence linking serum 25 hydroxy-vitamin D (25(OH)D) concentrations to outcome in cardiovascular and other diseases. We have studied patients with acute myocardial infarction (AMI) to determine if they exhibit an acute phase reaction affecting 25(OH)D.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Methods<\/jats:title>\n                    <jats:p>Patients ( n= 32) with first AMI who had been treated with primary percutaneous coronary intervention within 12 h of symptom onset had venous blood samples taken two days, one week, one month and three months after presentation. Samples were analysed for troponin I, C-reactive protein (CRP) and 25(OH)D.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Results<\/jats:title>\n                    <jats:p>All patients had significant rises in troponin confirming the myocardial damage and CRP, both of which resolved by 28 days. In contrast, 25(OH)D remained unchanged throughout the 90-day observation period with a median concentration of 46 nmol\/L.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Conclusion<\/jats:title>\n                    <jats:p>Serum 25(OH)D does not change after AMI and is likely to be a reliable marker of vitamin D status in patients with cardiovascular disease.<\/jats:p>\n                  <\/jats:sec>","DOI":"10.1258\/acb.2011.011195","type":"journal-article","created":{"date-parts":[[2012,4,28]],"date-time":"2012-04-28T00:40:55Z","timestamp":1335573655000},"page":"399-401","update-policy":"https:\/\/doi.org\/10.1177\/sage-journals-update-policy","source":"Crossref","is-referenced-by-count":21,"title":["Serum 25 hydroxy-vitamin D does not exhibit an acute phase reaction after acute myocardial infarction"],"prefix":"10.1177","volume":"49","author":[{"given":"J H","family":"Barth","sequence":"first","affiliation":[{"name":"Clinical Biochemistry, Leeds General Infirmary, Leeds LS1 3EX"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"H P","family":"Field","sequence":"additional","affiliation":[{"name":"Clinical Biochemistry, Leeds General Infirmary, Leeds LS1 3EX"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"A N","family":"Mather","sequence":"additional","affiliation":[{"name":"LIGHT Institute and Multidisciplinary Cardiovascular Research Centre, University of Leeds, Leeds, UK"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"S","family":"Plein","sequence":"additional","affiliation":[{"name":"LIGHT Institute and Multidisciplinary Cardiovascular Research Centre, University of Leeds, Leeds, UK"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"179","published-online":{"date-parts":[[2012,4,27]]},"reference":[{"key":"bibr-ACB-11-195C1","doi-asserted-by":"publisher","DOI":"10.2215\/CJN.02260409"},{"key":"bibr-ACB-11-195C2","doi-asserted-by":"publisher","DOI":"10.1359\/jbmr.090819"},{"key":"bibr-ACB-11-195C3","first-page":"1675","volume":"21","author":"Guha C","year":"1995","journal-title":"Hepatology"},{"key":"bibr-ACB-11-195C4","doi-asserted-by":"publisher","DOI":"10.1097\/00003246-199802000-00027"},{"key":"bibr-ACB-11-195C5","doi-asserted-by":"publisher","DOI":"10.1016\/j.trstmh.2005.06.022"},{"key":"bibr-ACB-11-195C6","doi-asserted-by":"publisher","DOI":"10.1093\/ajcn\/88.2.582S"},{"key":"bibr-ACB-11-195C7","doi-asserted-by":"publisher","DOI":"10.3945\/ajcn.110.008490"}],"container-title":["Annals of Clinical Biochemistry: International Journal of Laboratory Medicine"],"original-title":[],"language":"en","link":[{"URL":"https:\/\/journals.sagepub.com\/doi\/pdf\/10.1258\/acb.2011.011195","content-type":"application\/pdf","content-version":"vor","intended-application":"text-mining"},{"URL":"https:\/\/journals.sagepub.com\/doi\/full-xml\/10.1258\/acb.2011.011195","content-type":"application\/xml","content-version":"vor","intended-application":"text-mining"},{"URL":"https:\/\/journals.sagepub.com\/doi\/pdf\/10.1258\/acb.2011.011195","content-type":"unspecified","content-version":"vor","intended-application":"similarity-checking"}],"deposited":{"date-parts":[[2026,4,28]],"date-time":"2026-04-28T11:56:49Z","timestamp":1777377409000},"score":1,"resource":{"primary":{"URL":"https:\/\/journals.sagepub.com\/doi\/10.1258\/acb.2011.011195"}},"subtitle":[],"short-title":[],"issued":{"date-parts":[[2012,4,27]]},"references-count":7,"journal-issue":{"issue":"4","published-print":{"date-parts":[[2012,7]]}},"alternative-id":["10.1258\/acb.2011.011195"],"URL":"https:\/\/doi.org\/10.1258\/acb.2011.011195","relation":{},"ISSN":["0004-5632","1758-1001"],"issn-type":[{"value":"0004-5632","type":"print"},{"value":"1758-1001","type":"electronic"}],"subject":[],"published":{"date-parts":[[2012,4,27]]}}}