{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2024,6,20]],"date-time":"2024-06-20T20:10:16Z","timestamp":1718914216796},"reference-count":23,"publisher":"Walter de Gruyter GmbH","issue":"2-3","license":[{"start":{"date-parts":[[2016,11,1]],"date-time":"2016-11-01T00:00:00Z","timestamp":1477958400000},"content-version":"unspecified","delay-in-days":0,"URL":"http:\/\/creativecommons.org\/licenses\/by-nc-nd\/3.0"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":[],"published-print":{"date-parts":[[2016,11,1]]},"abstract":"<jats:title>Abstract<\/jats:title><jats:p>Regular ultrasound examinations carried out in the second trimester of pregnancy help in detecting many anomalies in the fetal urinary tract. Their percentage ranges from 1% to 3% of all controlled pregnancies. There is a wide spectrum of anomalies that affect the urinary tract, but the most significant are: uretero\/hydronephrosis (unilateral or bilateral), kidney agenesis, dysplastic kidney, polycystic and multicystic kidneys, anomalies of ascent, anomalies of kidney rotation or fusion, bladder exstrophy, posterior urethra valve etc.<\/jats:p><jats:p>Many of these anomalies do not have impact either on urine flow or on kidney function and hence they can be qualified rather as a condition than as a disease. At the same time, most of the hydronephroses that are seen prenatally are being resolved spontaneously, and they are not detected neither presented postnatally as uretero\/hydronephroses of unobstructed type and do not require surgical treatment. Only one tenth of these anomalies are subject to active surgical treatment.<\/jats:p><jats:p>Therefore, the assessment of these conditions should be done by a specialized team, who will make adequate therapeutic decisions based on clinical guidelines, as well as will advise the parents on the future clinical implications of the detected anomaly.<\/jats:p>","DOI":"10.1515\/prilozi-2016-0023","type":"journal-article","created":{"date-parts":[[2016,11,24]],"date-time":"2016-11-24T08:23:37Z","timestamp":1479975817000},"page":"107-113","source":"Crossref","is-referenced-by-count":0,"title":["Diagnostic and Surgical Approach to Prenatally Detected Urinary Tract Anomalies"],"prefix":"10.1515","volume":"37","author":[{"given":"Mile","family":"Petrovski","sequence":"first","affiliation":[{"name":"University Clinic of Pediatric Surgery, Medical Faculty, Skopje, Macedonia (the former Yugoslav Republic of)"}],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"374","published-online":{"date-parts":[[2016,11,23]]},"reference":[{"key":"2021040914364768529_j_prilozi-2016-0023_ref_001_w2aab2b8c25b1b7b1ab1ab1Aa","unstructured":"1. 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J Pediatr Surg. 1994; 29: 769\u201372."},{"key":"2021040914364768529_j_prilozi-2016-0023_ref_005_w2aab2b8c25b1b7b1ab1ab5Aa","doi-asserted-by":"crossref","unstructured":"5. Shokeir AA, Nijman RJM. Antenatal hydronephrosis: changing concepts in diagnosis and subsequent managemnet. BJU Int. 2000; 85: 987\u201394.","DOI":"10.1046\/j.1464-410x.2000.00645.x"},{"key":"2021040914364768529_j_prilozi-2016-0023_ref_006_w2aab2b8c25b1b7b1ab1ab6Aa","doi-asserted-by":"crossref","unstructured":"6. Grignon A, Filiatrault D, Hoinsy Y. Uretero pelvic junction stenosis, antenatal ultrasonographic diagnosis, postnatal investigation and follow up. Radiology. 1986; 160: 649\u201351.","DOI":"10.1148\/radiology.160.3.3526403"},{"key":"2021040914364768529_j_prilozi-2016-0023_ref_007_w2aab2b8c25b1b7b1ab1ab7Aa","doi-asserted-by":"crossref","unstructured":"7. Blyth B, Snyder HM, Ducket JF. Antenatal hydronephrosis and subsequent managemnet. 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Ureteral obstruction in the neonatal guinea pig: interaction of sympathetic nerves and angiotensin. Pediatr Nephrol. 1995; 9: 441\u20136.","DOI":"10.1007\/BF00866723"},{"key":"2021040914364768529_j_prilozi-2016-0023_ref_017_w2aab2b8c25b1b7b1ab1ac17Aa","doi-asserted-by":"crossref","unstructured":"17. Yanagisawa H, Morrissey J, Morrison AR, Klahr H. Eicosanoid production by isolated glomeruli of rats with unilateral ureteral obstruction. Kidney Int. 1990; 37: 1528\u201335.","DOI":"10.1038\/ki.1990.145"},{"key":"2021040914364768529_j_prilozi-2016-0023_ref_018_w2aab2b8c25b1b7b1ab1ac18Aa","doi-asserted-by":"crossref","unstructured":"18. Schreiner GF, Harris KP, Purkerson ML, Klahr S. Immunological aspects of acute ureteral obstruction: immune cell infiltrate in the kidney. Kidney Int. 1988; 34: 487.","DOI":"10.1038\/ki.1988.207"},{"key":"2021040914364768529_j_prilozi-2016-0023_ref_019_w2aab2b8c25b1b7b1ab1ac19Aa","doi-asserted-by":"crossref","unstructured":"19. Kennedy WA, Buttyan R, Sawczuk IS, et al. Epidermal growth factor suppresses renal tubular apoptosis following ureteral obstruction. Urology. 1997; 49: 973\u201380.","DOI":"10.1016\/S0090-4295(97)00101-5"},{"key":"2021040914364768529_j_prilozi-2016-0023_ref_020_w2aab2b8c25b1b7b1ab1ac20Aa","unstructured":"20. Restrepo N, Kess-Folts D, Ding G. Tubulointerstitial macrophages, transforming growth factorbeta, and monocyte chemoattractant peptide (MCP)-1 after unilateral ureteral obstruction. J Am Soc Nephrol. 1993; 4: 826\u201333."},{"key":"2021040914364768529_j_prilozi-2016-0023_ref_021_w2aab2b8c25b1b7b1ab1ac21Aa","unstructured":"21. Carr MC. Anomalies and surgery of the ureteropelvic junction in children. In: Walsh PC, Retik BA, Vaughan ED, Wein AJ, editors. Campbell\u2019s Urology. 8th ed. CD-ROM. Philadelphia: Saunders; 2003."},{"key":"2021040914364768529_j_prilozi-2016-0023_ref_022_w2aab2b8c25b1b7b1ab1ac22Aa","doi-asserted-by":"crossref","unstructured":"22. Sinha A, Bagga A, Krishna A, Bajpai M, Srinivas M, et al. Revised Guidelines on Management of Antenatal Hydronephrosis. Indian Pediatr. 2013; 50: 215\u201331.","DOI":"10.1007\/s13312-013-0064-6"},{"key":"2021040914364768529_j_prilozi-2016-0023_ref_023_w2aab2b8c25b1b7b1ab1ac23Aa","doi-asserted-by":"crossref","unstructured":"23. Kirwan D. NHS Fetal Anomaly Screening Programme. 18 to 20 Weeks Fetal Anomaly Scan National Standards and Guidance for England. 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