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We therefore studied axonal crossing with a functional approach, based on magnetoencephalography, in 10 dyslexic individuals who all share the same rare, weakly expressing haplotype of the<jats:italic>ROBO1<\/jats:italic>gene. Auditory-cortex responses were recorded separately to left- and right-ear sounds that were amplitude modulated at different frequencies. We found impaired interaural interaction that depended on the<jats:italic>ROBO1<\/jats:italic>in a dose-dependent manner. Our results indicate that normal crossing of the auditory pathways requires an adequate<jats:italic>ROBO1<\/jats:italic>expression level.<\/jats:p>","DOI":"10.1523\/jneurosci.4007-11.2012","type":"journal-article","created":{"date-parts":[[2012,1,18]],"date-time":"2012-01-18T19:02:25Z","timestamp":1326913345000},"page":"966-971","source":"Crossref","is-referenced-by-count":51,"title":["Human<i>ROBO1<\/i>Regulates Interaural Interaction in Auditory 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