{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2023,11,2]],"date-time":"2023-11-02T11:20:13Z","timestamp":1698924013377},"reference-count":44,"publisher":"Akademiai Kiado Zrt.","issue":"22","license":[{"start":{"date-parts":[[2021,5,30]],"date-time":"2021-05-30T00:00:00Z","timestamp":1622332800000},"content-version":"unspecified","delay-in-days":0,"URL":"http:\/\/creativecommons.org\/licenses\/by\/4.0\/"}],"content-domain":{"domain":["akjournals.com"],"crossmark-restriction":true},"short-container-title":["OH"],"published-print":{"date-parts":[[2021,5,30]]},"abstract":"<jats:p><jats:bold>\u00d6sszefoglal\u00f3.<\/jats:bold> A l\u00e1t\u00f3szerv k\u00fcl\u00f6nb\u00f6z\u0151 betegs\u00e9gei, valamint egyes\n                    sziszt\u00e9m\u00e1s megbeteged\u00e9sek r\u00e9szben vagy kifejezetten az idegh\u00e1rtya k\u00e1rosod\u00e1s\u00e1val\n                    j\u00e1rnak. A patol\u00f3gia seg\u00edts\u00e9g\u00e9vel ma m\u00e1r tudjuk, hogy ezek a betegs\u00e9gek a retina\n                    mely r\u00e9teg\u00e9nek vagy r\u00e9tegeinek elv\u00e1ltoz\u00e1sait okozz\u00e1k: m\u00edg az id\u0151skori\n                    maculadegeneratio a k\u00fcls\u0151 retin\u00e1ban tal\u00e1lhat\u00f3 fotoreceptorokat \u00e9rinti\n                    kifejezetten a fovea centralis ter\u00fclet\u00e9n, addig a glaucoma a bels\u0151 retina\n                    ganglionsejtjeinek pusztul\u00e1s\u00e1val, valamint e sejtek opticusrostjainak\n                    k\u00e1rosod\u00e1s\u00e1val j\u00e1r a stratum ganglionar\u00e9ban \u00e9s a stratum neurofibrarumban. Az\n                    emberi retina sejtjei azonban egyel\u0151re nem marad\u00e9ktalanul karakteriz\u00e1ltak, az\n                    egyes sejtt\u00edpusok sz\u00e1m\u00e1t csak becs\u00fclni tudjuk, \u00edgy nem \u00edrhat\u00f3k le az egyes\n                    sejtszint\u0171 elv\u00e1ltoz\u00e1sok sem kell\u0151 pontoss\u00e1ggal. A sz\u00f6vettani feldolgoz\u00e1s \u00e9s\n                    vizsg\u00e1lat megfelel\u0151 r\u00e9szletess\u00e9ggel t\u00e1j\u00e9koztat a diagn\u00f3zisr\u00f3l \u00e9s az elv\u00e1ltoz\u00e1s\n                    s\u00falyoss\u00e1g\u00e1r\u00f3l, \u00e9rtelemszer\u0171en azonban ez a m\u00f3dszer <jats:italic>in vivo<\/jats:italic> nem\n                    haszn\u00e1lhat\u00f3 a mindennapi klinikai gyakorlatban. A sejtszint\u0171 elv\u00e1ltoz\u00e1sok\n                    ismerete az egyes k\u00f3rk\u00e9pekben felvetette \u00e9s sz\u00fcks\u00e9gess\u00e9 tette olyan <jats:italic>in\n                        vivo,<\/jats:italic> a klinikumban is alkalmazhat\u00f3 vizsg\u00e1l\u00f3m\u00f3dszerek\n                    kifejleszt\u00e9s\u00e9t, amelyek lehet\u0151s\u00e9get ny\u00fajtanak a retina neur\u00e1lis \u00e9s egy\u00e9b\n                    sejtjeinek cellul\u00e1ris \u00e9s szubcellul\u00e1ris szint\u0171 vizsg\u00e1lat\u00e1ra, ide\u00e9rtve a v\u00e9r\n                    alakos elemeit is, amelyek eg\u00e9szs\u00e9ges vagy neovascularis eredet\u0171 erekben\n                    \u00e1ramlanak. A jelenleg is haszn\u00e1lt klinikai vizsg\u00e1latok mellett ezek a k\u00e9palkot\u00f3\n                    m\u00f3dszerek seg\u00edts\u00e9g\u00fcl szolg\u00e1lhatnak a diagn\u00f3zis meger\u0151s\u00edt\u00e9s\u00e9ben vagy elvet\u00e9s\u00e9ben,\n                    emellett az elv\u00e1ltoz\u00e1s s\u00falyoss\u00e1g\u00e1nak meg\u00edt\u00e9l\u00e9s\u00e9ben, valamint a progresszi\u00f3 vagy\n                    remisszi\u00f3 monitoroz\u00e1s\u00e1ban. Orv Hetil. 2021; 162(22): 851\u2013860.<\/jats:p>\n                <jats:p><jats:bold>Summary.<\/jats:bold> Diseases of the visual system as well as many systemic\n                    illnesses are usually associated with retinal damage. With the help of\n                    pathology, we can clearly identify the affected layer(s): while age-related\n                    macular degeneration mostly damages the photoreceptors in the outer retina at\n                    the central fovea, glaucoma promotes ganglion cell death in the ganglion cell\n                    layer and damages respective neural fibers. However, the diverse cell types of\n                    the human retina have not been fully characterized yet, thus in most cases our\n                    knowledge on cellular pathologies is not precise enough. While histopathological\n                    preparation and examination of the retinal tissue provide more detailed\n                    information about the diagnosis and the severity of the condition,\n                    unfortunately, it cannot be used <jats:italic>in vivo<\/jats:italic> in everyday clinical\n                    practice. Our understanding of the cellular changes in different diseases has\n                    revealed a need for new everyday clinical examination methods that can be used\n                        <jats:italic>in vivo<\/jats:italic> to asses cellular and subcellular changes in neural\n                    and other cells of the retina, such as blood cells flowing in healthy vessels or\n                    in vessels of neovascular origin. In addition to the currently used clinical\n                    examination methods, these imaging methods could help confirm or dismiss\n                    diagnoses, assess the severity of a condition, and monitor disease progression\n                    or remission. Orv Hetil. 2021; 162(22): 851\u2013860.<\/jats:p>","DOI":"10.1556\/650.2021.32101","type":"journal-article","created":{"date-parts":[[2021,8,31]],"date-time":"2021-08-31T10:03:58Z","timestamp":1630404238000},"page":"851-860","update-policy":"http:\/\/dx.doi.org\/10.1556\/policypage.crossmark.crossref.1","source":"Crossref","is-referenced-by-count":1,"title":["Sejtszint\u0171 k\u00e9palkot\u00e1s a retina in vivo vizsg\u00e1lat\u00e1ban: jelen \u00e9s j\u00f6v\u0151"],"prefix":"10.1556","volume":"162","author":[{"given":"Andr\u00e1s","family":"V\u00e9gh","sequence":"first","affiliation":[{"name":"1 Semmelweis Egyetem, \u00c1ltal\u00e1nos Orvostudom\u00e1nyi Kar, Anat\u00f3miai, Sz\u00f6vet- \u00e9s Fejl\u0151d\u00e9stani Int\u00e9zet, Budapest, T\u0171zolt\u00f3 u. 58., 1094"},{"name":"2 Semmelweis Egyetem, \u00c1ltal\u00e1nos Orvostudom\u00e1nyi Kar, Szem\u00e9szeti Klinika, Budapest"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"D\u00e1niel P\u00e9ter","family":"Magda","sequence":"additional","affiliation":[{"name":"1 Semmelweis Egyetem, \u00c1ltal\u00e1nos Orvostudom\u00e1nyi Kar, Anat\u00f3miai, Sz\u00f6vet- \u00e9s Fejl\u0151d\u00e9stani Int\u00e9zet, Budapest, T\u0171zolt\u00f3 u. 58., 1094"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Ferenc","family":"Kilin","sequence":"additional","affiliation":[{"name":"1 Semmelweis Egyetem, \u00c1ltal\u00e1nos Orvostudom\u00e1nyi Kar, Anat\u00f3miai, Sz\u00f6vet- \u00e9s Fejl\u0151d\u00e9stani Int\u00e9zet, Budapest, T\u0171zolt\u00f3 u. 58., 1094"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Anita","family":"Csorba","sequence":"additional","affiliation":[{"name":"2 Semmelweis Egyetem, \u00c1ltal\u00e1nos Orvostudom\u00e1nyi Kar, Szem\u00e9szeti Klinika, Budapest"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Mik\u00f3s","family":"Resch","sequence":"additional","affiliation":[{"name":"2 Semmelweis Egyetem, \u00c1ltal\u00e1nos Orvostudom\u00e1nyi Kar, Szem\u00e9szeti Klinika, Budapest"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Zolt\u00e1n Zsolt","family":"Nagy","sequence":"additional","affiliation":[{"name":"2 Semmelweis Egyetem, \u00c1ltal\u00e1nos Orvostudom\u00e1nyi Kar, Szem\u00e9szeti Klinika, Budapest"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Arnold","family":"Szab\u00f3","sequence":"additional","affiliation":[{"name":"1 Semmelweis Egyetem, \u00c1ltal\u00e1nos Orvostudom\u00e1nyi Kar, Anat\u00f3miai, Sz\u00f6vet- \u00e9s Fejl\u0151d\u00e9stani Int\u00e9zet, Budapest, T\u0171zolt\u00f3 u. 58., 1094"}],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"425","reference":[{"key":"ref0","unstructured":"1 Szab\u00f3 A. 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