{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,11,2]],"date-time":"2026-11-02T17:31:45Z","timestamp":1793640705220,"version":"4.5.1"},"reference-count":158,"publisher":null,"issue":"9","license":[{"start":{"date-parts":[[2019,9,21]],"date-time":"2019-09-21T00:00:00Z","timestamp":1569024000000},"content-version":"vor","delay-in-days":0,"URL":"https:\/\/lib.ossn.ru\/jour\/about\/editorialPolicies#openAccessPolicy"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Kardiologiia"],"abstract":"<jats:p>During several recent decades spontaneous coronary artery dissection (SCAD) has been known as one of causes of development\u00a0of acute coronary syndrome (ACS). It has been assumed that this condition is extremely rarely met and is associated with pregnancy\u00a0and postpartum period. The use in clinical practice of high sensitivity troponin, coronary angiography (CAG) in early period of ACS,\u00a0in conjunction with the growing awareness of doctors about this pathology led to a revision of the viewse on prevalence of the disease.\u00a0At present SCAD is considered as one of the causes of ACS in young and middle-aged women. In this review we present results\u00a0of studies of pathogenesis, diagnostics, and treatment of SCAD, describe various angiographic types of this disease, and discuss problems\u00a0of choice of optimal strategy of management of patients with SCAD.<\/jats:p>","DOI":"10.18087\/cardio.2019.9.10269","type":"journal-article","created":{"date-parts":[[2019,9,21]],"date-time":"2019-09-21T04:02:06Z","timestamp":1569038526000},"page":"52-63","source":"Crossref","is-referenced-by-count":13,"title":["Spontaneous Coronary Artery Dissection"],"prefix":"10.68900","volume":"59","author":[{"given":"N. S.","family":"Zhukova","sequence":"first","affiliation":[{"name":"National Medical Research Center for Cardiology"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"R. M.","family":"Shakhnovich","sequence":"additional","affiliation":[{"name":"National Medical Research Center for Cardiology"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"I. N.","family":"Merkulova","sequence":"additional","affiliation":[{"name":"National Medical Research Center for Cardiology"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"T. S.","family":"Sukhinina","sequence":"additional","affiliation":[{"name":"National Medical Research Center for Cardiology"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"D. V.","family":"Pevzner","sequence":"additional","affiliation":[{"name":"National Medical Research Center for Cardiology"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"I. I.","family":"Staroverov","sequence":"additional","affiliation":[{"name":"National Medical Research Center for Cardiology"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":null,"published-online":{"date-parts":[[2019,9,21]]},"reference":[{"key":"ref1","doi-asserted-by":"crossref","unstructured":"Robinowitz M, Virmani R, McAllister HA JrU null. Spontaneous coronary artery dissection and eosinophilic inflammation: a cause and effect relationship? The American Journal of Medicine. 1982;72(6):923\u20138. PMID: 7091163","DOI":"10.1016\/0002-9343(82)90853-1"},{"key":"ref2","doi-asserted-by":"crossref","unstructured":"Saw J, Mancini GBJ, Humphries KH. Contemporary Review on Spontaneous Coronary Artery Dissection. Journal of the American College of Cardiology. 2016;68(3):297\u2013312. DOI: 10.1016\/j.jacc.2016.05.034","DOI":"10.1016\/j.jacc.2016.05.034"},{"key":"ref3","doi-asserted-by":"crossref","unstructured":"Bulkley BH, Roberts WC. 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