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Pure red cell aplasia associated with thymic follicular hyperplasia. Saudi Med J 28: 798-799, 2007."},{"key":"6","doi-asserted-by":"crossref","unstructured":"6. Mohammad A, Dawson AG, Bajaj A, Rathinam S. True thymic hyperplasia causing pure red cell aplasia: a case report. Interact Cardiovasc Thorac Surg 34: 697-699, 2022.","DOI":"10.1093\/icvts\/ivab301"},{"key":"7","doi-asserted-by":"crossref","unstructured":"7. Kabir A, Alizadehfar R, Tsoukas CM. Good&apos;s syndrome: time to move on from reviewing the past. Front Immunol 12: 815710, 2021.","DOI":"10.3389\/fimmu.2021.815710"},{"key":"8","doi-asserted-by":"crossref","unstructured":"8. Yamada Y, Tomonaga M, Fukuda H, et al. A new G-CSF-supported combination chemotherapy, LSG15, for adult T-cell leukaemia-lymphoma: Japan Clinical Oncology Group Study 9303. Br J Haematol 113: 375-382, 2001.","DOI":"10.1046\/j.1365-2141.2001.02737.x"},{"key":"9","doi-asserted-by":"crossref","unstructured":"9. Malphettes M, G\u00e9rard L, Galicier L, et al. Good syndrome: an adult-onset immunodeficiency remarkable for its high incidence of invasive infections and autoimmune complications. Clin Infect Dis 61: e13-e19, 2015.","DOI":"10.1093\/cid\/civ269"},{"key":"10","doi-asserted-by":"crossref","unstructured":"10. Tarr PE, Sneller MC, Mechanic LJ, et al. Infections in patients with immunodeficiency with thymoma (Good syndrome). Report of 5 cases and review of the literature. Medicine (Baltimore) 80: 123-133, 2001.","DOI":"10.1097\/00005792-200103000-00005"},{"key":"11","doi-asserted-by":"crossref","unstructured":"11. Morgan E, Pang KM, Goldwasser E. Hodgkin disease and red cell aplasia. Am J Hematol 5: 71-75, 1978.","DOI":"10.1002\/ajh.2830050110"},{"key":"12","doi-asserted-by":"crossref","unstructured":"12. Narra K, Borghaei H, Al-Saleem T, H\u00f6glund M, Smith MR. Pure red cell aplasia in B-cell lymphoproliferative disorder treated with rituximab: report of two cases and review of the literature. Leuk Res 30: 109-114, 2006.","DOI":"10.1016\/j.leukres.2005.05.017"},{"key":"13","doi-asserted-by":"crossref","unstructured":"13. Tsujimura H, Sakai C, Takagi T. Pure red cell aplasia complicated by angioimmunoblastic T-cell lymphoma: humoral factor plays a main role in the inhibition of erythropoiesis from CD34<sup>+<\/sup> progenitor cells. Am J Hematol 62: 259-260, 1999.","DOI":"10.1002\/(SICI)1096-8652(199912)62:4<259::AID-AJH13>3.0.CO;2-5"},{"key":"14","doi-asserted-by":"crossref","unstructured":"14. Hirokawa M, Sawada K, Fujishima N, et al. Acquired pure red cell aplasia associated with malignant lymphomas: a nationwide cohort study in Japan for the PRCA Collaborative Study Group. Am J Hematol 84: 144-148, 2009.","DOI":"10.1002\/ajh.21354"},{"key":"15","doi-asserted-by":"crossref","unstructured":"15. Kawakami F, Kawakami T, Yamane T, et al. T cell clonal expansion and <i>STAT3<\/i> mutations: a characteristic feature of acquired chronic T cell-mediated pure red cell aplasia. Int J Hematol 115: 816-825, 2022.","DOI":"10.1007\/s12185-022-03310-2"},{"key":"16","doi-asserted-by":"crossref","unstructured":"16. Koskela HL, Eldfors S, Ellonen P, et al. Somatic <i>STAT3<\/i> mutations in large granular lymphocytic leukemia. N Engl J Med 366: 1905-1913, 2012.","DOI":"10.1056\/NEJMoa1114885"},{"key":"17","doi-asserted-by":"crossref","unstructured":"17. Ishida F, Matsuda K, Sekiguchi N, et al. <i>STAT3<\/i> gene mutations and their association with pure red cell aplasia in large granular lymphocyte leukemia. Cancer Sci 105: 342-346, 2014.","DOI":"10.1111\/cas.12341"},{"key":"18","doi-asserted-by":"crossref","unstructured":"18. Teramo A, Baril\u00e0 G, Calabretto G, et al. <i>STAT3<\/i> mutation impacts biological and clinical features of T-LGL leukemia. 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