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Arafat AM, M\u00f6hlig M, Weickert MO, et al. Growth hormone response during oral glucose tolerance test: the impact of assay method on the estimation of reference values in patients with acromegaly and in healthy controls, and the role of gender, age, and body mass index. J Clin Endocrinol Metab 93: 1254-1262, 2008.","DOI":"10.1210\/jc.2007-2084"},{"key":"2","doi-asserted-by":"crossref","unstructured":"2. Hage M, Kamenick\u00fd P, Chanson P. Growth hormone response to oral glucose load: from normal to pathological conditions. Neuroendocrinology 108: 244-255, 2019.","DOI":"10.1159\/000497214"},{"key":"3","doi-asserted-by":"crossref","unstructured":"3. Melmed S. Acromegaly pathogenesis and treatment. J Clin Invest 119: 3189-3202, 2009.","DOI":"10.1172\/JCI39375"},{"key":"4","doi-asserted-by":"crossref","unstructured":"4. Melmed S. Acromegaly. N Engl J Med 322: 966-977, 1990.","DOI":"10.1056\/NEJM199004053221405"},{"key":"5","doi-asserted-by":"crossref","unstructured":"5. Umahara M, Okada S, Ohshima K, Mori M. Glucose-dependent insulinotropic polypeptide induced growth hormone secretion in acromegaly. Endocr J 50: 643-650, 2003.","DOI":"10.1507\/endocrj.50.643"},{"key":"6","doi-asserted-by":"crossref","unstructured":"6. Occhi G, Losa M, Albiger N, et al. The glucose-dependent insulinotropic polypeptide receptor is overexpressed amongst GNAS1 mutation-negative somatotropinomas and drives growth hormone (GH)-promoter activity in GH3 cells. J Neuroendocrinol 23: 641-649, 2011.","DOI":"10.1111\/j.1365-2826.2011.02155.x"},{"key":"7","doi-asserted-by":"crossref","unstructured":"7. Chanson P, Salenave S, Kamenicky P, Cazabat L, Young J. Pituitary tumours: acromegaly. Best Pract Res Clin Endocrinol Metab 23: 555-574, 2009.","DOI":"10.1016\/j.beem.2009.05.010"},{"key":"8","doi-asserted-by":"crossref","unstructured":"8. Katznelson L, Laws ER, Jr., Melmed S, et al. Acromegaly: an endocrine society clinical practice guideline. 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Pituitary 17: 81-89, 2014.","DOI":"10.1007\/s11102-013-0471-7"},{"key":"12","doi-asserted-by":"crossref","unstructured":"12. Fieffe S, Morange I, Petrossians P, et al. Diabetes in acromegaly, prevalence, risk factors, and evolution: data from the French Acromegaly Registry. Eur J Endocrinol 164: 877-884, 2011.","DOI":"10.1530\/EJE-10-1050"},{"key":"13","doi-asserted-by":"crossref","unstructured":"13. Dreval AV, Trigolosova IV, Misnikova IV, et al. Prevalence of diabetes mellitus in patients with acromegaly. Endocr Connect 3: 93-98, 2014.","DOI":"10.1530\/EC-14-0021"},{"key":"14","doi-asserted-by":"crossref","unstructured":"14. Gadelha MR, Kasuki L, Lim DST, Fleseriu M. Systemic complications of acromegaly and the impact of the current treatment landscape: an update. Endocr Rev 40: 268-332, 2019.","DOI":"10.1210\/er.2018-00115"},{"key":"15","doi-asserted-by":"crossref","unstructured":"15. Peracchi M, Porretti S, Gebbia C, et al. Increased glucose-dependent insulinotropic polypeptide (GIP) secretion in acromegaly. Eur J Endocrinol 145: R1-4, 2001.","DOI":"10.1530\/eje.0.145r001"},{"key":"16","doi-asserted-by":"crossref","unstructured":"16. Shekhawat VS, Bhansali S, Dutta P, et al. Glucose-dependent insulinotropic polypeptide (GIP) resistance and \u03b2-cell dysfunction contribute to hyperglycaemia in acromegaly. Sci Rep 9: 5646, 2019.","DOI":"10.1038\/s41598-019-41887-7"},{"key":"17","doi-asserted-by":"crossref","unstructured":"17. Herman GA, Stevens C, Van Dyck K, et al. Pharmacokinetics and pharmacodynamics of sitagliptin, an inhibitor of dipeptidyl peptidase IV, in healthy subjects: results from two randomized, double-blind, placebo-controlled studies with single oral doses. Clin Pharmacol Ther 78: 675-688, 2005.","DOI":"10.1016\/j.clpt.2005.09.002"},{"key":"18","doi-asserted-by":"crossref","unstructured":"18. Volante M, Brizzi MP, Faggiano A, et al. Somatostatin receptor type 2A immunohistochemistry in neuroendocrine tumors: a proposal of scoring system correlated with somatostatin receptor scintigraphy. Mod Pathol 20: 1172-1182, 2007.","DOI":"10.1038\/modpathol.3800954"},{"key":"19","doi-asserted-by":"crossref","unstructured":"19. Hage M, Chalign\u00e9 R, Viengchareun S, et al. Hypermethylator phenotype and ectopic GIP receptor in GNAS mutation-negative somatotropinomas. J Clin Endocrinol Metab 104: 1777-1787, 2019.","DOI":"10.1210\/jc.2018-01504"},{"key":"20","doi-asserted-by":"crossref","unstructured":"20. Hage M, Viengchareun S, Brunet E, et al. Genomic alterations and complex subclonal architecture in sporadic GH-secreting pituitary adenomas. J Clin Endocrinol Metab 103: 1929-1939, 2018.","DOI":"10.1210\/jc.2017-02287"},{"key":"21","doi-asserted-by":"crossref","unstructured":"21. Regazzo D, Losa M, Albiger NM, et al. The GIP\/GIPR axis is functionally linked to GH-secretion increase in a significant proportion of gsp<sup>-<\/sup> somatotropinomas. Eur J Endocrinol 176: 543-553, 2017.","DOI":"10.1530\/EJE-16-0831"},{"key":"22","doi-asserted-by":"publisher","unstructured":"22. Klemann C, Wagner L, Stephan M, von Horsten S. Cut to the chase: a review of CD26\/dipeptidyl peptidase-4&apos;s (DPP4) entanglement in the immune system. Clin Exp Immunol 185: 1-21, 2016.","DOI":"10.1111\/cei.12781"},{"key":"23","doi-asserted-by":"crossref","unstructured":"23. Grecu EO, Walter RM, Jr., Gold EM. Paradoxical release of growth hormone during oral glucose tolerance test in patients with abnormal glucose tolerance. 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