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Tashima K, Ando Y, Terazaki H, et al. Outcome of liver transplantation for transthyretin amyloidosis: follow-up of Japanese familial amyloidotic polyneuropathy patients. J Neurol Sci 171: 19-23, 1999.","DOI":"10.1016\/S0022-510X(99)00231-2"},{"key":"2","doi-asserted-by":"publisher","unstructured":"2. Yamashita T, Ando Y, Okamoto S, et al. Long-term survival after liver transplantation in patients with familial amyloid polyneuropathy. Neurology 78: 637-643, 2012.","DOI":"10.1212\/WNL.0b013e318248df18"},{"key":"3","doi-asserted-by":"crossref","unstructured":"3. Ericzon BG, Wilczek H, Larsson M, et al. Liver transplantation for hereditary transthyretin amyloidosis: after 20 years still the best therapeutic alternative? Transplantation 99: 1847-1854, 2015.","DOI":"10.1097\/TP.0000000000000574"},{"key":"4","doi-asserted-by":"crossref","unstructured":"4. Saeki Y, Ueno S, Yorifuji S, Sugiyama Y, Ide Y, Matsuzawa Y. New mutant gene (transthyretin Arg 58) in cases with hereditary polyneuropathy detected by non-isotope method of single-strand conformation polymorphism analysis. Biochem Biophys Res Commun 180: 380-385, 1991.","DOI":"10.1016\/S0006-291X(05)81304-X"},{"key":"5","doi-asserted-by":"crossref","unstructured":"5. Motozaki Y, Sugiyama Y, Ishida C, Komai K, Matsubara S, Yamada M. Phenotypic heterogeneity in a family with FAP due to a TTR Leu58Arg mutation: a clinicopathologic study. J Neurol Sci 260: 236-239, 2007.","DOI":"10.1016\/j.jns.2007.03.021"},{"key":"6","doi-asserted-by":"crossref","unstructured":"6. Kato-Motozaki Y, Ono K, Shima K, et al. Epidemiology of familial amyloid polyneuropathy in Japan: identification of a novel endemic focus. J Neurol Sci 270: 133-140, 2008.","DOI":"10.1016\/j.jns.2008.02.019"},{"key":"7","doi-asserted-by":"crossref","unstructured":"7. Bergstr\u00f6m J, Gustavsson \u00c5, Hellman U, et al. Amyloid deposits in transthyretin-derived amyloidosis: cleaved transthyretin is associated with distinct amyloid morphology. J Pathol 206: 224-232, 2005.","DOI":"10.1002\/path.1759"},{"key":"8","doi-asserted-by":"crossref","unstructured":"8. Ihse E, Rapezzi C, Merlini G, et al. Amyloid fibrils containing fragmented ATTR may be the standard fibril compostion in ATTR amyloidosis. Amyloid 20: 142-150, 2013.","DOI":"10.3109\/13506129.2013.797890"},{"key":"9","doi-asserted-by":"crossref","unstructured":"9. Koike H, Ando Y, Ueda M, et al. Distinct characteristics of amyloid deposits in early- and late-onset transthyretin Val30Met familial amyloid polyneuropathy. J Neurol Sci 287: 178-184, 2009.","DOI":"10.1016\/j.jns.2009.07.028"},{"key":"10","unstructured":"10. Nakanuma S, Takamura H, Shoji M, et al. Case report of an ABO-incompatible living-donor liver transplant for a familial amyloid polyneuropathy patient. Exp Clin Transplant 13: 479-481, 2015."},{"key":"11","doi-asserted-by":"publisher","unstructured":"11. Gillmore JD, Maurer MS, Falk RH, et al. Nonbiopsy diagnosis of cardiac transthyretin amyloidosis. Circulation 133: 2404-2412, 2016.","DOI":"10.1161\/CIRCULATIONAHA.116.021612"},{"key":"12","doi-asserted-by":"crossref","unstructured":"12. Sekijima Y. Transthyretin (ATTR) amyloidosis: clinical spectrum, molecular pathogenesis and disease-modifying treatments. J Neurol Neurosurg Psychiatry 86: 1036-1043, 2015.","DOI":"10.1136\/jnnp-2014-308724"},{"key":"13","doi-asserted-by":"crossref","unstructured":"13. Finsterer J, Iglseder S, Wanschitz J, Topakian R, L\u00f6scher WN, Grisold W. Hereditary transthyretin-related amyloidosis. Acta Neurol Scand 139: 92-105, 2019.","DOI":"10.1111\/ane.13035"},{"key":"14","doi-asserted-by":"publisher","unstructured":"14. Okumura K, Yamashita T, Masuda T, et al. Long-term outcome of patients with hereditary transthyretin V30M amyloidosis with polyneuropathy after liver transplantation. Amyloid 23: 39-45, 2016.","DOI":"10.3109\/13506129.2015.1123149"},{"key":"15","doi-asserted-by":"crossref","unstructured":"15. Kon T, Misumi Y, Nishijima H, et al. Effects of liver transplantation and tafamidis in hereditary transthyretin amyloidosis caused by transthyretin Leu55Pro mutation: a case report. Amyloid 22: 203-204, 2015.","DOI":"10.3109\/13506129.2015.1031373"},{"key":"16","doi-asserted-by":"crossref","unstructured":"16. Muller KR, Padbury R, Jeffrey GP, et al. Poor outcome after liver transplantation for transthyretin amyloid neuropathy in a family with an Ala36Pro transthyretin mutation: case report. Liver Transpl 16: 470-473, 2010.","DOI":"10.1002\/lt.22019"},{"key":"17","doi-asserted-by":"publisher","unstructured":"17. Suhr OB, Larsson M, Ericzon BG, Wilczek HE; FAPWTR's investigators. Survival after transplantation in patients with mutations other than Val30Met: extracts from the FAP World Transplant Registry. Transplantation 100: 373-381, 2016.","DOI":"10.1097\/TP.0000000000001021"},{"key":"18","doi-asserted-by":"crossref","unstructured":"18. Moshe-Lilie O, Dimitrova D, Heitner SB, et al. TTR gene silencing therapy in post liver transplant hereditary ATTR amyloidosis patients. Amyloid 27: 250-253, 2020.","DOI":"10.1080\/13506129.2020.1784134"},{"key":"19","doi-asserted-by":"crossref","unstructured":"19. Yazaki M, Mitsuhashi S, Tokuda T, et al. Progressive wild-type transthyretin deposition after liver transplantation preferentially occurs onto myocardium in FAP patients. Am J Transplant 7: 235-242, 2007.","DOI":"10.1111\/j.1600-6143.2006.01585.x"},{"key":"20","doi-asserted-by":"crossref","unstructured":"20. Liepnieks JJ, Zhang LQ, Benson MD. Progression of transthyrein amyloid neuropathy after liver transplantation. Neurology 75: 324-327, 2010.","DOI":"10.1212\/WNL.0b013e3181ea15d4"},{"key":"21","doi-asserted-by":"crossref","unstructured":"21. Koike H, Nishi R, Ikeda S, et al. The morphology of amyloid fibrils and their impact on tissue damages in hereditary transthyretin amyloidosis: an ultrastructural study. J Neurol Sci 394: 99-106, 2018.","DOI":"10.1016\/j.jns.2018.09.011"},{"key":"22","doi-asserted-by":"crossref","unstructured":"22. Gustafsson S, Ihse E, Henein MY, Westermark P, Lindqvist P, Suhr OB. Amyloid fibril composition as a predictor of development of cardiomyopathy after liver transplantation for hereditary transthyretin amyloidosis. 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