{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,10,14]],"date-time":"2026-10-14T11:41:26Z","timestamp":1791978086215,"version":"4.4.0"},"reference-count":25,"publisher":"American Diabetes Association","issue":"8","license":[{"start":{"date-parts":[[2026,6,8]],"date-time":"2026-06-08T00:00:00Z","timestamp":1780876800000},"content-version":"vor","delay-in-days":0,"URL":"https:\/\/www.diabetesjournals.org\/journals\/pages\/license"}],"funder":[{"DOI":"10.13039\/100004326","name":"Bayer AG","doi-asserted-by":"crossref","id":[{"id":"10.13039\/100004326","id-type":"DOI","asserted-by":"crossref"}]}],"content-domain":{"domain":["diabetesjournals.org"],"crossmark-restriction":true},"short-container-title":[],"published-print":{"date-parts":[[2026,8,1]]},"abstract":"<jats:sec>\n                    <jats:title>OBJECTIVE<\/jats:title>\n                    <jats:p>To evaluate whether the efficacy and safety of finerenone varied by baseline hemoglobin A1c (HbA1c) level, a proxy of glycemic control, and diabetes duration in people with type 1 diabetes and chronic kidney disease (CKD).<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>RESEARCH DESIGN AND METHODS<\/jats:title>\n                    <jats:p>Adults with type 1 diabetes, urinary albumin-to-creatinine ratio (UACR) 200 to &amp;lt;5,000 mg\/g, and estimated glomerular filtration rate (eGFR) 25 to &amp;lt;90 mL\/min\/1.73 m2 were randomly assigned (one to one) to finerenone or placebo. UACR change from baseline over 6 months by baseline HbA1c and diabetes duration was analyzed.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>RESULTS<\/jats:title>\n                    <jats:p>Baseline HbA1c was available for 240 of 242 participants; mean (SD) HbA1c, diabetes duration, and eGFR were 7.6% (1.1%; 60 [12] mmol\/mol), 32.0 (14.2) years, and 58.9 (19.2) mL\/min\/1.73 m2, respectively. At 6 months, HbA1c (95% CI) remained unchanged (finerenone +0.03% [\u22120.14%, 0.20%]; placebo 0.00% [\u22120.12%, 0.11%]; between-group difference +0.04% [\u22120.17%, 0.24%]; P = 0.74). Over 6 months, median UACR decreased from 574.6 to 373.5 mg\/g with finerenone and from 506.4 to 475.6 mg\/g with placebo, corresponding to a \u221225% placebo-corrected change (95% CI \u201335%, \u201313%; P = 0.0001). Treatment effects were consistent across HbA1c tertiles (&amp;lt;7.1%, \u22657.1% to \u22648.1%, and &amp;gt;8.1%), with placebo-corrected UACR changes (95% CIs) of \u221217% (\u221240%, 13%), \u221218% (\u221239%, 10%), and \u221237% (\u221255%, \u221213%), respectively (P interaction = 0.41). Effects were similarly consistent across diabetes duration tertiles (P interaction = 0.70). Overall safety and incidence of hyperkalemia were similar across HbA1c tertiles.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>CONCLUSIONS<\/jats:title>\n                    <jats:p>In adults with type 1 diabetes and CKD, finerenone reduced UACR and was well tolerated irrespective of HbA1c level or diabetes duration.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title\/>\n                  <\/jats:sec>","DOI":"10.2337\/dc26-0882","type":"journal-article","created":{"date-parts":[[2026,6,8]],"date-time":"2026-06-08T17:31:40Z","timestamp":1780939900000},"page":"1434-1441","update-policy":"https:\/\/doi.org\/10.2337\/ada-journal-policies","source":"Crossref","is-referenced-by-count":1,"title":["Effect of Finerenone on Albuminuria in Type 1 Diabetes by Baseline HbA1c Level and Diabetes Duration: An Exploratory Analysis of the FINE-ONE Trial"],"prefix":"10.2337","volume":"49","author":[{"ORCID":"https:\/\/orcid.org\/0000-0002-4615-9012","authenticated-orcid":false,"given":"Jelle M.","family":"Beernink","sequence":"first","affiliation":[{"name":"Department of Clinical Pharmacy and Pharmacology, University of Groningen, University Medical Center Groningen 1 , Groningen, the Netherlands"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"ORCID":"https:\/\/orcid.org\/0000-0002-3126-3730","authenticated-orcid":false,"given":"Hiddo J.L.","family":"Heerspink","sequence":"additional","affiliation":[{"name":"Department of Clinical Pharmacy and Pharmacology, University of Groningen, University Medical Center Groningen 1 , Groningen, the Netherlands"},{"name":"The George Institute for Global Health, University of New South Wales 2 , Sydney, Australia"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"ORCID":"https:\/\/orcid.org\/0000-0003-1407-9023","authenticated-orcid":false,"given":"Andreas L.","family":"Birkenfeld","sequence":"additional","affiliation":[{"name":"Institute for Diabetes Research and Metabolic Diseases, Helmholtz Centre Munich, University of T\u00fcbingen 3 , T\u00fcbingen, Germany"},{"name":"German Center for Diabetes Research 4 , Neuherberg, Germany"},{"name":"Internal Medicine IV, Department of Endocrinology, Diabetology and Nephrology, University of T\u00fcbingen 5 , T\u00fcbingen, Germany"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"ORCID":"https:\/\/orcid.org\/0000-0003-4164-0429","authenticated-orcid":false,"given":"David Z.I.","family":"Cherney","sequence":"additional","affiliation":[{"name":"Division of Nephrology, University Health Network, Toronto General Hospital, University of Toronto 6 , Toronto, Ontario, Canada"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"ORCID":"https:\/\/orcid.org\/0000-0002-8345-3288","authenticated-orcid":false,"given":"Helen 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