{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,2,14]],"date-time":"2026-02-14T07:19:54Z","timestamp":1771053594000,"version":"3.50.1"},"reference-count":36,"publisher":"Journal of Neurosurgery Publishing Group (JNSPG)","issue":"2","content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":[],"published-print":{"date-parts":[[1974,8]]},"abstract":"<jats:p content-type=\"fine-print\">\u2713 Six cases of bilateral hemorrhage into the vitreous body related to intracranial hypertension are presented. Four were associated with ruptured cerebral aneurysms, and the others followed head injury. The onset of vitreous hemorrhage was delayed in all cases, and in five patients subhyaloid hemorrhages were present from 2 to 27 days prior to their extension into the vitreous. Visual acuity was greatly reduced. The ophthalmoscopic and slit lamp appearance of the vitreous are described. The hemorrhages usually cleared spontaneously within 24 months, and vision returned to normal. Surgical treatment to remove residual vitreous blood in selected cases is outlined.<\/jats:p>","DOI":"10.3171\/jns.1974.41.2.0167","type":"journal-article","created":{"date-parts":[[2009,5,7]],"date-time":"2009-05-07T15:00:46Z","timestamp":1241708446000},"page":"167-176","source":"Crossref","is-referenced-by-count":62,"title":["Vitreous hemorrhages and sudden increased intracranial pressure"],"prefix":"10.3171","volume":"41","author":[{"given":"R. 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