{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,10,1]],"date-time":"2026-10-01T12:20:37Z","timestamp":1790857237810,"version":"4.1.0"},"reference-count":22,"publisher":"Journal of Neurosurgery Publishing Group (JNSPG)","issue":"6","content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":[],"published-print":{"date-parts":[[1978,6]]},"abstract":"<jats:p content-type=\"fine-print\">\u2713 The early sequential histopathological alterations following a concussive paraplegic injury to the posterior thoracic spinal cord in cats were studied. The lack of significant progression of hemorrhages over a 4-hour period after injury indicates that most hemorrhages probably occur within the first hour. The marked enhancement or retardation of hemorrhages in the post-injury period, when the blood pressure was increased or decreased, respectively, demonstrates the loss of autoregulation of spinal cord vasculature at the trauma site after a concussive paraplegic injury. Progressive edema formation was evident over a 4-hour period following injury, and it could be enhanced or retarded by elevation or reduction of the systemic blood pressure.<\/jats:p>","DOI":"10.3171\/jns.1978.48.6.1002","type":"journal-article","created":{"date-parts":[[2009,5,7]],"date-time":"2009-05-07T15:31:27Z","timestamp":1241710287000},"page":"1002-1007","source":"Crossref","is-referenced-by-count":34,"title":["The histopathology of experimental spinal cord trauma"],"prefix":"10.3171","volume":"48","author":[{"given":"Stephen E.","family":"Rawe","sequence":"first","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"William A.","family":"Lee","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Phanor L.","family":"Perot","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"1754","reference":[{"key":"b1","series-title":"Radiology","doi-asserted-by":"publisher","first-page":"107","DOI":"10.1148\/111.1.107","article-title":"Correlation of microangiographic and electrophysiologic changes in experimental spinal cord trauma.","volume":"111","author":"Allen","year":"1974"},{"key":"b2","series-title":"Br J Anaesth","doi-asserted-by":"publisher","first-page":"927","DOI":"10.1093\/bja\/39.12.927","article-title":"Cerebral vasodilatation by halothane anaesthesia in man and its potentiation by hypotension and hypercapnia.","volume":"39","author":"Christensen","year":"1967"},{"key":"b3","series-title":"J Neurosurg","doi-asserted-by":"publisher","first-page":"332","DOI":"10.3171\/jns.1973.38.3.0332","article-title":"Evoked cortical potentials in experimental spinal cord trauma.","volume":"38","author":"D'Angelo","year":"1973"},{"key":"b4","series-title":"J Trauma","doi-asserted-by":"publisher","first-page":"1003","DOI":"10.1097\/00005373-197511000-00011","article-title":"Microcirculation of traumatized spinal cord. 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