{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2024,9,8]],"date-time":"2024-09-08T11:35:48Z","timestamp":1725795348624},"reference-count":15,"publisher":"Journal of Neurosurgery Publishing Group (JNSPG)","issue":"4","content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":[],"published-print":{"date-parts":[[1989,10]]},"abstract":"<jats:p content-type=\"fine-print\">\u2713 Sodium nitroprusside is commonly used for the induction of hypotension during neurosurgical procedures. Its toxicity stems from hemodynamic compromise as well as from its metabolites, especially the formation of cyanide. A patient is described who underwent craniotomy for hypertensive intracerebral hemorrhage. He gradually recovered following the operation, but needed continued administration of sodium nitroprusside for control of hypertension. On the 7th postoperative day, he deteriorated into coma with evidence of severe edema and herniation on the computerized tomography scan. Cessation of sodium nitroprusside and treatment for cyanide poisoning resulted in resolution of his symptoms within hours. The potential toxicity of sodium nitroprusside, measures to prevent toxicity, and therapeutic steps are discussed.<\/jats:p>","DOI":"10.3171\/jns.1989.71.4.0605","type":"journal-article","created":{"date-parts":[[2009,5,8]],"date-time":"2009-05-08T16:23:22Z","timestamp":1241799802000},"page":"605-607","source":"Crossref","is-referenced-by-count":10,"title":["Delayed postoperative neurological deterioration from prolonged sodium nitroprusside administration"],"prefix":"10.3171","volume":"71","author":[{"given":"Zvi","family":"Ram","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Roberto","family":"Spiegelman","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Gideon","family":"Findler","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Moshe","family":"Hadani","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"1754","reference":[{"key":"b1","series-title":"Neurosurgery","doi-asserted-by":"publisher","first-page":"50","DOI":"10.1227\/00006123-197807000-00008","article-title":"Effect of intravenous sodium nitroprusside on cerebral blood flow and intracranial pressure.","volume":"3","author":"Candia","year":"1978"},{"key":"b2","series-title":"J Neurosurg","doi-asserted-by":"publisher","first-page":"329","DOI":"10.3171\/jns.1978.48.3.0329","article-title":"Intracranial pressure changes induced by sodium nitroprusside in patients with intracranial mass lesions.","volume":"48","author":"Cottrell","year":"1978"},{"key":"b3","series-title":"Can Anaesth Soc J","doi-asserted-by":"publisher","first-page":"547","DOI":"10.1007\/BF03013407","article-title":"A sudden death associated with the use of sodium nitroprusside for induction of hypotension during anesthesia.","volume":"22","author":"Davies","year":"1975"},{"key":"b4","series-title":"Medical Toxicology. 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