{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2025,10,10]],"date-time":"2025-10-10T12:43:06Z","timestamp":1760100186179},"reference-count":32,"publisher":"Journal of Neurosurgery Publishing Group (JNSPG)","issue":"3","content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":[],"published-print":{"date-parts":[[1993,3]]},"abstract":"<jats:p content-type=\"fine-print\">\u2713 Percutaneous transluminal angioplasty for treatment of cerebral vasospasm was performed in primates. Chronic cerebral vasospasm was induced by placement of an autologous blood clot over the right internal carotid artery (ICA), middle cerebral artery (MCA), and anterior cerebral artery (ACA). Cerebral angiography on Day 7 showed that the diameters of the ICA, MCA, and ACA were reduced to 55.7% \u00b1 1.3%, 55.3% \u00b1 2.6%, and 59.6% \u00b1 1.3%, respectively, of baseline. The angioplasty was carried out with a silicone microballoon attached to a microcatheter under somatosensory evoked potential (SEP) monitoring on Day 7. The angioplasty for ICA was performed satisfactorily; however, the balloon could be not advanced to the spastic M<jats:sub>1<\/jats:sub> or A<jats:sub>1<\/jats:sub> portions of the cerebral artery. Following angioplasty, the diameters of the ICA, the M<jats:sub>1<\/jats:sub> segment, and the A<jats:sub>1<\/jats:sub> segment were 79.6% \u00b1 2.9% (p &lt; 0.001), 67.6% \u00b1 4.3% (p &lt; 0.05), and 61.7% \u00b1 2.2% (not significant), respectively, of baseline. Histological studies demonstrated that the vessels were well dilated and patent without endothelial cell damage.<\/jats:p>","DOI":"10.3171\/jns.1993.78.3.0481","type":"journal-article","created":{"date-parts":[[2009,5,27]],"date-time":"2009-05-27T15:30:56Z","timestamp":1243438256000},"page":"481-486","source":"Crossref","is-referenced-by-count":37,"title":["Histological studies of intracranial vessels in primates following transluminal angioplasty for vasospasm"],"prefix":"10.3171","volume":"78","author":[{"given":"Hidenori","family":"Kobayashi","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Hisashi","family":"Ide","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Hiroshi","family":"Aradachi","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Yoshikazu","family":"Arai","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Yuji","family":"Handa","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Toshihiko","family":"Kubota","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"1754","reference":[{"key":"b1","series-title":"Exp Neurol","first-page":"592","article-title":"A comparative analysis of short-latency somatosensory evoked potentials in man, monkey, cat, and rat.","volume":"72","author":"Allison","year":"1981"},{"key":"b2","series-title":"Stroke","first-page":"365","article-title":"Clinical vasospasm after subarachnoid hemorrhage: response to hypervolemic hemodilution and arterial hypertension.","volume":"18","author":"Awad","year":"1987"},{"key":"b3","series-title":"AJNR","first-page":"239","article-title":"Intracranial angioplasty for treatment of vasospasm after subarachnoid hemorrhage: technique and modifications to improve branch access.","volume":"11","author":"Brothers","year":"1990"},{"key":"b4","series-title":"Neurol Res","first-page":"12","article-title":"Morphological changes in normal canine basilar arteries after transluminal angioplasty.","volume":"12","author":"Chavez","year":"1990"},{"key":"b5","series-title":"Circulation","first-page":"654","article-title":"Transluminal treatment of arteriosclerotic obstruction. 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