{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,10,10]],"date-time":"2026-10-10T02:15:04Z","timestamp":1791598504790,"version":"4.3.4"},"reference-count":21,"publisher":"SAGE Publications","issue":"2-3","license":[{"start":{"date-parts":[[2019,6,12]],"date-time":"2019-06-12T00:00:00Z","timestamp":1560297600000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/journals.sagepub.com\/page\/policies\/text-and-data-mining-license"}],"content-domain":{"domain":["journals.sagepub.com"],"crossmark-restriction":true},"short-container-title":["Journal of Vestibular Research"],"published-print":{"date-parts":[[2019,7,25]]},"abstract":"<jats:sec specific-use=\"heading-level-1\">\n                    <jats:title>BACKGROUND\/OBJECTIVES:<\/jats:title>\n                    <jats:p>Vestibular paroxysmia (VP) presents as episodic vertigo believed to be caused by neurovascular cross-compression (NVCC) of the vestibulocochlear nerve. We investigated whether NVCC occurred at a higher rate in VP, compared with controls and whether angulation of the nerve, the vessel involved and location of the point of contact were significant features.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec specific-use=\"heading-level-1\">\n                    <jats:title>METHODS:<\/jats:title>\n                    <jats:p>Retrospective analysis was conducted of MR imaging performed in patients with VP and also patients with unilateral tinnitus (in whom the asymptomatic side was used as a control). Two independent, blinded reviewers assessed each case.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec specific-use=\"heading-level-1\">\n                    <jats:title>RESULTS:<\/jats:title>\n                    <jats:p>\n                      Nine patients with VP and 20 patients with unilateral tinnitus were included. NVCC was demonstrated in all 9 VP patients (100%), compared with 9 of the controls (45%),\n                      <jats:italic>p<\/jats:italic>\n                      \u200a=\u200a0.0049. NVCC was mostly caused by a branch of the anterior inferior cerebellar artery (AICA). Nerve angulation at the point of contact occurred in 5 of the cases (44%), but in none of the controls (specificity\u200a=\u200a100%),\n                      <jats:italic>p<\/jats:italic>\n                      \u200a=\u200a0.0053. There was no correlation between site of contact and VP.\n                    <\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec specific-use=\"heading-level-1\">\n                    <jats:title>CONCLUSION:<\/jats:title>\n                    <jats:p>Our study supports the concept of NVCC in VP and additionally suggests that nerve angulation may be a specific feature. Neurovascular contact remains a common phenomenon in asymptomatic patients and therefore correlation with neuro-otology assessment remains essential.<\/jats:p>\n                  <\/jats:sec>","DOI":"10.3233\/ves-190661","type":"journal-article","created":{"date-parts":[[2019,7,26]],"date-time":"2019-07-26T12:29:29Z","timestamp":1564144169000},"page":"137-145","update-policy":"https:\/\/doi.org\/10.1177\/sage-journals-update-policy","source":"Crossref","is-referenced-by-count":29,"title":["MRI findings in vestibular paroxysmia - An observational study"],"prefix":"10.1177","volume":"29","author":[{"given":"Nishanth","family":"Sivarasan","sequence":"first","affiliation":[{"name":"Department of Radiology, Guy\u2019s and St Thomas\u2019 NHS Foundation Trust, London, UK"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Philip","family":"Touska","sequence":"additional","affiliation":[{"name":"Department of Radiology, Guy\u2019s and St Thomas\u2019 NHS Foundation Trust, London, UK"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Louisa","family":"Murdin","sequence":"additional","affiliation":[{"name":"Department of ENT, Guy\u2019s and St Thomas\u2019 NHS Foundation Trust, London, UK"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Steve","family":"Connor","sequence":"additional","affiliation":[{"name":"Department of Radiology, King\u2019s College Hospital NHS Foundation Trust, London, UK"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"179","published-online":{"date-parts":[[2019,6,12]]},"reference":[{"key":"e_1_3_1_2_2","doi-asserted-by":"publisher","DOI":"10.1016\/j.pain.2014.04.020"},{"key":"e_1_3_1_3_2","doi-asserted-by":"publisher","DOI":"10.3174\/ajnr.A5156"},{"key":"e_1_3_1_4_2","doi-asserted-by":"publisher","DOI":"10.1136\/jnnp-2013-305513"},{"key":"e_1_3_1_5_2","first-page":"565","article-title":"VIIIth nerve vascular compression syndrome: Vestibular paroxysmia","volume":"3","author":"Brandt T.","year":"1994","unstructured":"BrandtT., DieterichM., VIIIth nerve vascular compression syndrome: Vestibular paroxysmia, Baillieres Clin Neurol 3 (1994), 565\u201375.","journal-title":"Baillieres Clin Neurol"},{"key":"e_1_3_1_6_2","doi-asserted-by":"publisher","DOI":"10.1227\/01.NEU.0000186035.73828.34"},{"key":"e_1_3_1_7_2","doi-asserted-by":"publisher","DOI":"10.1016\/j.rcl.2016.08.001"},{"key":"e_1_3_1_8_2","doi-asserted-by":"publisher","DOI":"10.1007\/s00701-011-1168-1"},{"key":"e_1_3_1_9_2","doi-asserted-by":"publisher","DOI":"10.3174\/ajnr.A1212"},{"key":"e_1_3_1_10_2","doi-asserted-by":"publisher","DOI":"10.3174\/ajnr.A4683"},{"key":"e_1_3_1_11_2","doi-asserted-by":"crossref","unstructured":"H\u00fcfnerK. 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