{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,8,18]],"date-time":"2026-08-18T16:39:55Z","timestamp":1787071195747,"version":"build-2736575974"},"reference-count":11,"publisher":"Ovid Technologies (Wolters Kluwer Health)","issue":"6","content-domain":{"domain":["lww.com","ovid.com"],"crossmark-restriction":true},"short-container-title":[],"published-print":{"date-parts":[[2014,11]]},"abstract":"\n Background:<\/jats:title>\n \n Reactive arthritis (ReA) is defined as a peripheral arthritis lasting longer than 1 month, associated with urethritis, cervicitis, or diarrhea. The reported annual incidence of ReA is approximately 30-40 cases per 100,000 adults, occurring commonly in the age group of 16 and 35 years. It is known to be associated with gastrointestinal infections with\n Shigella, Salmonella<\/jats:italic>\n , and\n Campylobacter<\/jats:italic>\n species and other microorganisms, as well as with genitourinary infections (especially with\n Chlamydia trachomatis<\/jats:italic>\n ).\n <\/jats:p>\n <\/jats:sec>\n \n Case Report:<\/jats:title>\n This article reports the case of a 53-year-old, post-right total hip replacement, Indian man, with ReA, who presented with fever, respiratory distress, and abdominal discomfort. He complained of itching, tingling sensation, pain on urination, and retention of urine. He had right hip joint pain for 3 weeks, inability to move right leg since 10 days, and melena since 1 week. Laboratory tests revealed anemia, high liver and kidney function tests, elevated erythrocyte sedimentation rate, C reactive protein, procalcitonin and occult blood in stool. He tested positive for hepatitis C virus genotype 3. Gastroduodenoscopy revealed multiple apthoid ulcers at D2 and large gastric varix. Ultrasonography of whole abdomen revealed cholelithiasis and splenomegaly. Skin lesions and arthritis led to the diagnosis of associated ReA. The patient was managed conservatively and discharged in a stable condition.<\/jats:p>\n <\/jats:sec>\n \n Conclusions:<\/jats:title>\n Our case is unlike classical ReA because the patient is older, HLA B27 negative, and without florid urethritis. Admitted for fever and lower urinary tract symptoms, along with respiratory distress, the primary objective of the emergency doctors was to prevent the patient from progressing to organ failure. The diagnosis of underlying atypical\/incomplete ReA could easily have been missed without adequate awareness, dermatological consultation, and a skin biopsy.<\/jats:p>\n <\/jats:sec>","DOI":"10.4103\/0019-5154.143524","type":"journal-article","created":{"date-parts":[[2014,10,29]],"date-time":"2014-10-29T03:05:15Z","timestamp":1414551915000},"page":"584-587","update-policy":"https:\/\/doi.org\/10.1097\/lww.0000000000001000","source":"Crossref","is-referenced-by-count":0,"title":["Portal Hypertension and an Atypical Reactive Arthritis Like Presentation in a patient infected with Hepatitis C Virus Genotype 3"],"prefix":"10.4103","volume":"59","author":[{"given":"Moushumi","family":"Lodh","sequence":"first","affiliation":[{"name":"From the Department of Biochemistry, The Mission Hospital, Durgapur, West Bengal, India"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Md Ashif Ali","family":"Ahmed","sequence":"additional","affiliation":[{"name":"Department of Gastroenterology, The Mission Hospital, Durgapur, West Bengal, India"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Bidisha Guha","family":"Niyogi","sequence":"additional","affiliation":[{"name":"Department of Dermatology, The Mission Hospital, Durgapur, West Bengal, India"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Brinda","family":"Bandyopadhyay","sequence":"additional","affiliation":[{"name":"Department of Gastroenterology, The Mission Hospital, Durgapur, West Bengal, India"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"276","reference":[{"key":"R1-20250921","doi-asserted-by":"crossref","first-page":"348","DOI":"10.1128\/CMR.17.2.348-369.2004","article-title":"HLA-B27-associated reactive arthritis: Pathogenetic and clinical considerations","volume":"17","author":"Colmegna","year":"2004","journal-title":"Clin Microbiol Rev"},{"key":"R2-20250921","doi-asserted-by":"crossref","first-page":"386","DOI":"10.1097\/BOR.0b013e32832aac66","article-title":"Epidemiologic approaches to infection and immunity: The case of reactive arthritis","volume":"21","author":"Rohekar","year":"2009","journal-title":"Curr Opin Rheumatol"},{"key":"R3-20250921","doi-asserted-by":"crossref","first-page":"11","DOI":"10.1007\/s10067-008-0968-z","article-title":"Reactive and undifferentiated arthritis in North Africa: Use of PCR for detection of Chlamydia trachomatis","volume":"28","author":"Kuipers","year":"2009","journal-title":"Clin Rheumatol"},{"key":"R4-20250921","first-page":"197","article-title":"Reactive arthritis","volume":"59","author":"Madavamurthy","year":"1993","journal-title":"Indian J Dermatol Venereol Leprol"},{"key":"R6-20250921","doi-asserted-by":"crossref","first-page":"677","DOI":"10.1016\/S0889-857X(05)70037-5","article-title":"Reiter's syndrome. 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