{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,10,21]],"date-time":"2026-10-21T22:22:40Z","timestamp":1792621360361,"version":"4.5.0"},"reference-count":159,"publisher":"Ovid Technologies (Wolters Kluwer Health)","issue":"2","content-domain":{"domain":["lww.com","ovid.com"],"crossmark-restriction":true},"short-container-title":[],"published-print":{"date-parts":[[2021,3]]},"abstract":"<jats:sec>\n                    <jats:title\/>\n                    <jats:p>Involuntary movements develop after 1\u20134% of strokes and they have been reported in patients with ischemic and hemorrhagic strokes affecting the basal ganglia, thalamus, and\/or their connections. Hemichorea-hemiballism is the most common movement disorder following a stroke in adults while dystonia is most common in children. Tremor, myoclonus, asterixis, stereotypies, and vascular parkinsonism are other movement disorders seen following stroke. Some of them occur immediately after acute stroke, some can develop later, and others may have delayed onset progressive course. Proposed pathophysiological mechanisms include neuronal plasticity, functional diaschisis, and age-related differences in brain metabolism. There are no guidelines regarding the management of post-stroke movement disorders, mainly because of their heterogeneity.<\/jats:p>\n                  <\/jats:sec>","DOI":"10.4103\/0028-3886.314574","type":"journal-article","created":{"date-parts":[[2021,4,26]],"date-time":"2021-04-26T01:30:50Z","timestamp":1619400650000},"page":"272-283","update-policy":"https:\/\/doi.org\/10.1097\/lww.0000000000001000","source":"Crossref","is-referenced-by-count":136,"title":["Post-stroke Movement Disorders"],"prefix":"10.4103","volume":"69","author":[{"given":"Priyanka","family":"Tater","sequence":"first","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Sanjay","family":"Pandey","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"276","reference":[{"key":"R1-6-20240907","doi-asserted-by":"crossref","first-page":"109","DOI":"10.1016\/S0022-510X(96)00290-0","article-title":"Hyperkinetic movement disorders during and after acute stroke: The Lausanne Stroke Registry","volume":"146","author":"Ghika-Schmid","year":"1997","journal-title":"J Neurol Sci"},{"key":"R2-6-20240907","doi-asserted-by":"crossref","first-page":"1568","DOI":"10.1136\/jnnp.2003.011874","article-title":"Post-stroke movement disorders: Report of 56 patients","volume":"75","author":"Alarc\u00f3n","year":"2004","journal-title":"J Neurol Neurosurg Psychiatry"},{"key":"R3-6-20240907","doi-asserted-by":"crossref","first-page":"260","DOI":"10.1093\/ageing\/afp020","article-title":"Movement disorders after stroke","volume":"38","author":"Handley","year":"2009","journal-title":"Age Ageing"},{"key":"R4-6-20240907","doi-asserted-by":"crossref","first-page":"59","DOI":"10.1136\/jnnp.72.1.59","article-title":"The natural history and treatment of acquired hemidystonia: Report of 33 cases and review of the literature","volume":"72","author":"Chuang","year":"2002","journal-title":"J Neurol Neurosurg Psychiatry"},{"key":"R5-6-20240907","doi-asserted-by":"crossref","first-page":"299","DOI":"10.1093\/brain\/124.2.299","article-title":"Delayed onset mixed involuntary movements after thalamic stroke: Clinical, radiological and pathophysiological findings","volume":"124","author":"Kim","year":"2001","journal-title":"Brain J Neurol"},{"key":"R6-6-20240907","doi-asserted-by":"crossref","first-page":"597","DOI":"10.1016\/S1474-4422(13)70057-7","article-title":"Movement disorders in cerebrovascular disease","volume":"12","author":"Mehanna","year":"2013","journal-title":"Lancet Neurol"},{"key":"R7-6-20240907","first-page":"42","article-title":"Movement disorders after stroke in adults: A review","volume":"2","author":"Bansil","year":"2012","journal-title":"Tremor Other Hyperkinet Mov (N Y"},{"key":"R8-6-20240907","doi-asserted-by":"crossref","first-page":"71","DOI":"10.14802\/jmd.16005","article-title":"Movement disorders following cerebrovascular lesion in the Basal Ganglia Circuit","volume":"9","author":"Park","year":"2016","journal-title":"J Mov Disord"},{"key":"R10-6-20240907","doi-asserted-by":"crossref","first-page":"254","DOI":"10.2174\/157015912803217341","article-title":"Post-stroke movement disorders: Clinical manifestations and pharmacological management","volume":"10","author":"Siniscalchi","year":"2012","journal-title":"Curr Neuropharmacol"},{"key":"R11-6-20240907","doi-asserted-by":"crossref","first-page":"351","DOI":"10.1001\/archneur.60.3.351","article-title":"Acute infarction limited to the lenticular nucleus: Clinical, etiologic, and topographic features","volume":"60","author":"Russmann","year":"2003","journal-title":"Arch Neurol"},{"key":"R12-6-20240907","doi-asserted-by":"crossref","first-page":"485","DOI":"10.1093\/brain\/108.2.485","article-title":"Nonhaemorrhagic thalamic infarction. 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