{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,8,14]],"date-time":"2026-08-14T20:27:11Z","timestamp":1786739231728,"version":"build-2736575974"},"reference-count":10,"publisher":"Ovid Technologies (Wolters Kluwer Health)","issue":"1","content-domain":{"domain":["lww.com","ovid.com"],"crossmark-restriction":true},"short-container-title":[],"published-print":{"date-parts":[[2021,1]]},"abstract":"<jats:sec>\n                    <jats:title\/>\n                    <jats:p>Congenital long QT syndrome (LQTS) is a rare cardiac condition characterized by abnormality of either sodium or potassium ion channels resulting in prolongation of QT interval and thereby predisposing to life-threatening arrhythmia. Once the syndrome is diagnosed, measures should be taken to avoid sudden cardiac death. We present a rare case of LQTS associated with patent ductus arteriosus in a child, and a unique approach was used in managing both conditions.<\/jats:p>\n                  <\/jats:sec>","DOI":"10.4103\/apc.apc_146_19","type":"journal-article","created":{"date-parts":[[2020,9,22]],"date-time":"2020-09-22T09:41:23Z","timestamp":1600767683000},"page":"85-87","update-policy":"https:\/\/doi.org\/10.1097\/lww.0000000000001000","source":"Crossref","is-referenced-by-count":1,"title":["Congenital long QT syndrome and patent ductus arteriosus"],"prefix":"10.4103","volume":"14","author":[{"given":"Javid","family":"Raja","sequence":"first","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Sabarinath","family":"Menon","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Devarakonda Bhargava","family":"Venkata","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"KP","family":"Unnikrishnan","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Narayanan","family":"Namboodiri","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"276","reference":[{"key":"R1-14-20240901","doi-asserted-by":"crossref","first-page":"783","DOI":"10.1161\/CIRCULATIONAHA.105.592899","article-title":"The Jervell and Lange-Nielsen syndrome: Natural history, molecular basis, and clinical outcome","volume":"113","author":"Schwartz","year":"2006","journal-title":"Circulation"},{"key":"R2-14-20240901","doi-asserted-by":"crossref","first-page":"1199","DOI":"10.1161\/01.CIR.0000157069.91834.DA","article-title":"25th Anniversary of the International Long-QT Syndrome Registry: An ongoing quest to uncover the secrets of long-QT syndrome","volume":"111","author":"Moss","year":"2005","journal-title":"Circulation"},{"key":"R3-14-20240901","doi-asserted-by":"crossref","first-page":"8089","DOI":"10.1073\/pnas.0502506102","article-title":"Severe arrhythmia disorder caused by cardiac L-type calcium channel mutations","volume":"103","author":"Splawski","year":"2005","journal-title":"Proc Natl Acad Sci USA"},{"key":"R4-14-20240901","doi-asserted-by":"crossref","first-page":"2929","DOI":"10.1161\/01.CIR.92.10.2929","article-title":"ECG T wave patterns in genetically distinct forms of the hereditary. 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